1R01AG027443/AG/NIA NIH HHS/United States[Grants and Funding] - Search Results

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Aβ oligomers inhibit synapse remodelling necessary for memory consolidation.

Freir DB, Fedriani R, Scully D, Smith IM, Selkoe DJ, Walsh DM, Regan CM. Freir DB, et al. Neurobiol Aging. 2011 Dec;32(12):2211-8. doi: 10.1016/j.neurobiolaging.2010.01.001. Epub 2010 Jan 25. Neurobiol Aging. 2011. PMID: 20097446 Free PMC article.

Biochemical and immunohistochemical analysis of an Alzheimer's disease mouse model reveals the presence of multiple cerebral Abeta assembly forms throughout life.

Shankar GM, Leissring MA, Adame A, Sun X, Spooner E, Masliah E, Selkoe DJ, Lemere CA, Walsh DM. Shankar GM, et al. Neurobiol Dis. 2009 Nov;36(2):293-302. doi: 10.1016/j.nbd.2009.07.021. Epub 2009 Aug 4. Neurobiol Dis. 2009. PMID: 19660551 Free PMC article.

Natural oligomers of the Alzheimer amyloid-beta protein induce reversible synapse loss by modulating an NMDA-type glutamate receptor-dependent signaling pathway.

Shankar GM, Bloodgood BL, Townsend M, Walsh DM, Selkoe DJ, Sabatini BL. Shankar GM, et al. J Neurosci. 2007 Mar 14;27(11):2866-75. doi: 10.1523/JNEUROSCI.4970-06.2007. J Neurosci. 2007. PMID: 17360908 Free PMC article.

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