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New mutations in the KVLQT1 potassium channel that cause long-QT syndrome.
Li H, Chen Q, Moss AJ, Robinson J, Goytia V, Perry JC, Vincent GM, Priori SG, Lehmann MH, Denfield SW, Duff D, Kaine S, Shimizu W, Schwartz PJ, Wang Q, Towbin JA. Li H, et al. Among authors: shimizu w. Circulation. 1998 Apr 7;97(13):1264-9. doi: 10.1161/01.cir.97.13.1264. Circulation. 1998. PMID: 9570196
Gene-specific response of dynamic ventricular repolarization to sympathetic stimulation in LQT1, LQT2 and LQT3 forms of congenital long QT syndrome.
Noda T, Takaki H, Kurita T, Suyama K, Nagaya N, Taguchi A, Aihara N, Kamakura S, Sunagawa K, Nakamura K, Ohe T, Horie M, Napolitano C, Towbin JA, Priori SG, Shimizu W. Noda T, et al. Among authors: shimizu w. Eur Heart J. 2002 Jun;23(12):975-83. doi: 10.1053/euhj.2001.3079. Eur Heart J. 2002. PMID: 12069453
Sympathetic stimulation produces a greater increase in both transmural and spatial dispersion of repolarization in LQT1 than LQT2 forms of congenital long QT syndrome.
Tanabe Y, Inagaki M, Kurita T, Nagaya N, Taguchi A, Suyama K, Aihara N, Kamakura S, Sunagawa K, Nakamura K, Ohe T, Towbin JA, Priori SG, Shimizu W. Tanabe Y, et al. Among authors: shimizu w. J Am Coll Cardiol. 2001 Mar 1;37(3):911-9. doi: 10.1016/s0735-1097(00)01200-6. J Am Coll Cardiol. 2001. PMID: 11693770 Free article.
899 results