Krieg T - Search Results

1

Health position paper and redox perspectives on reactive oxygen species as signals and targets of cardioprotection.

Heusch G, Andreadou I, Bell R, Bertero E, Botker HE, Davidson SM, Downey J, Eaton P, Ferdinandy P, Gersh BJ, Giacca M, Hausenloy DJ, Ibanez B, Krieg T, Maack C, Schulz R, Sellke F, Shah AM, Thiele H, Yellon DM, Di Lisa F. Heusch G, et al. Among authors: krieg t. Redox Biol. 2023 Nov;67:102894. doi: 10.1016/j.redox.2023.102894. Epub 2023 Oct 6. Redox Biol. 2023. PMID: 37839355 Free PMC article. Review.

2

Mitochondria and their role in preconditioning's trigger phase.

Krieg T, Cohen MV, Downey JM. Krieg T, et al. Basic Res Cardiol. 2003 Jul;98(4):228-34. doi: 10.1007/s00395-003-0422-y. Basic Res Cardiol. 2003. PMID: 12903644 Review. No abstract available.

3

P1075 opens mitochondrial K(ATP) channels and generates reactive oxygen species resulting in cardioprotection of rabbit hearts.

Oldenburg O, Yang XM, Krieg T, Garlid KD, Cohen MV, Grover GJ, Downey JM. Oldenburg O, et al. Among authors: krieg t. J Mol Cell Cardiol. 2003 Sep;35(9):1035-42. doi: 10.1016/s0022-2828(03)00151-2. J Mol Cell Cardiol. 2003. PMID: 12967626

4

Augmented taurine release is not the mechanism of ischemic preconditioning's cardioprotection.

Krieg T, Yang XM, Downey JM, Schaffer SW, Cohen MV. Krieg T, et al. Amino Acids. 2004 Jun;26(3):263-6. doi: 10.1007/s00726-003-0062-4. Epub 2004 Feb 4. Amino Acids. 2004. PMID: 15221506

5

Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by targeting cell signaling pathways.

Yang XM, Proctor JB, Cui L, Krieg T, Downey JM, Cohen MV. Yang XM, et al. Among authors: krieg t. J Am Coll Cardiol. 2004 Sep 1;44(5):1103-10. doi: 10.1016/j.jacc.2004.05.060. J Am Coll Cardiol. 2004. PMID: 15337225 Free article.

6

Mapping preconditioning's signaling pathways: an engineering approach.

Downey JM, Krieg T, Cohen MV. Downey JM, et al. Among authors: krieg t. Ann N Y Acad Sci. 2008 Mar;1123:187-96. doi: 10.1196/annals.1420.022. Ann N Y Acad Sci. 2008. PMID: 18375591

7

NECA and bradykinin at reperfusion reduce infarction in rabbit hearts by signaling through PI3K, ERK, and NO.

Yang XM, Krieg T, Cui L, Downey JM, Cohen MV. Yang XM, et al. Among authors: krieg t. J Mol Cell Cardiol. 2004 Mar;36(3):411-21. doi: 10.1016/j.yjmcc.2003.12.008. J Mol Cell Cardiol. 2004. PMID: 15010280

8

Mitochondrial ROS generation following acetylcholine-induced EGF receptor transactivation requires metalloproteinase cleavage of proHB-EGF.

Krieg T, Cui L, Qin Q, Cohen MV, Downey JM. Krieg T, et al. J Mol Cell Cardiol. 2004 Mar;36(3):435-43. doi: 10.1016/j.yjmcc.2003.12.013. J Mol Cell Cardiol. 2004. PMID: 15010282

9

Both A2a and A2b adenosine receptors at reperfusion are necessary to reduce infarct size in mouse hearts.

Methner C, Schmidt K, Cohen MV, Downey JM, Krieg T. Methner C, et al. Among authors: krieg t. Am J Physiol Heart Circ Physiol. 2010 Oct;299(4):H1262-4. doi: 10.1152/ajpheart.00181.2010. Epub 2010 Aug 13. Am J Physiol Heart Circ Physiol. 2010. PMID: 20709859 Free PMC article.

10

ACh and adenosine activate PI3-kinase in rabbit hearts through transactivation of receptor tyrosine kinases.

Krieg T, Qin Q, McIntosh EC, Cohen MV, Downey JM. Krieg T, et al. Am J Physiol Heart Circ Physiol. 2002 Dec;283(6):H2322-30. doi: 10.1152/ajpheart.00474.2002. Epub 2002 Aug 22. Am J Physiol Heart Circ Physiol. 2002. PMID: 12388234 Free article.

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