Lopez-Pedrera C - Search Results

1

Splicing machinery is impaired in rheumatoid arthritis, associated with disease activity and modulated by anti-TNF therapy.

Ibáñez-Costa A, Perez-Sanchez C, Patiño-Trives AM, Luque-Tevar M, Font P, Arias de la Rosa I, Roman-Rodriguez C, Abalos-Aguilera MC, Conde C, Gonzalez A, Pedraza-Arevalo S, Del Rio-Moreno M, Blazquez-Encinas R, Segui P, Calvo J, Ortega Castro R, Escudero-Contreras A, Barbarroja N, Aguirre MA, Castaño JP, Luque RM, Collantes-Estevez E, Lopez-Pedrera C. Ibáñez-Costa A, et al. Ann Rheum Dis. 2022 Jan;81(1):56-67. doi: 10.1136/annrheumdis-2021-220308. Epub 2021 Oct 8. Ann Rheum Dis. 2022. PMID: 34625402 Free PMC article.

2

[Pathogenic mechanisms of thrombosis in neoplasia: therapeutic implications].

López-Pedrera C, Barbarroja N, Velasco F. López-Pedrera C, et al. Med Clin (Barc). 2004 Feb 14;122(5):190-6. doi: 10.1016/s0025-7753(04)74189-5. Med Clin (Barc). 2004. PMID: 14998456 Review. Spanish.

3

Promyelocytic leukemia retinoid signaling targets regulate apoptosis,tissue factor and thrombomodulin expression.

López-Pedrera C, Barbarroja N, Buendía P, Torres A, Dorado G, Velasco F. López-Pedrera C, et al. Haematologica. 2004 Mar;89(3):286-95. Haematologica. 2004. PMID: 15020266

4

Antiphospholipid antibodies from patients with the antiphospholipid syndrome induce monocyte tissue factor expression through the simultaneous activation of NF-kappaB/Rel proteins via the p38 mitogen-activated protein kinase pathway, and of the MEK-1/ERK pathway.

López-Pedrera C, Buendía P, Cuadrado MJ, Siendones E, Aguirre MA, Barbarroja N, Montiel-Duarte C, Torres A, Khamashta M, Velasco F. López-Pedrera C, et al. Arthritis Rheum. 2006 Jan;54(1):301-11. doi: 10.1002/art.21549. Arthritis Rheum. 2006. PMID: 16385547 Free article.

5

Antiphospholipid-mediated thrombosis: interplay between anticardiolipin antibodies and vascular cells.

López-Pedrera C, Buendía P, Barbarroja N, Siendones E, Velasco F, Cuadrado MJ. López-Pedrera C, et al. Clin Appl Thromb Hemost. 2006 Jan;12(1):41-5. doi: 10.1177/107602960601200107. Clin Appl Thromb Hemost. 2006. PMID: 16444433 Free article. Review.

6

Proteomic analysis of acute myeloid leukemia: Identification of potential early biomarkers and therapeutic targets.

López-Pedrera C, Villalba JM, Siendones E, Barbarroja N, Gómez-Díaz C, Rodríguez-Ariza A, Buendía P, Torres A, Velasco F. López-Pedrera C, et al. Proteomics. 2006 Apr;6 Suppl 1:S293-9. doi: 10.1002/pmic.200500384. Proteomics. 2006. PMID: 16521150

7

Inhibition of Flt3-activating mutations does not prevent constitutive activation of ERK/Akt/STAT pathways in some AML cells: a possible cause for the limited effectiveness of monotherapy with small-molecule inhibitors.

Siendones E, Barbarroja N, Torres LA, Buendía P, Velasco F, Dorado G, Torres A, López-Pedrera C. Siendones E, et al. Hematol Oncol. 2007 Mar;25(1):30-7. doi: 10.1002/hon.805. Hematol Oncol. 2007. PMID: 17128418

8

Coordinated deregulation of cellular receptors, proangiogenic factors and intracellular pathways in acute myeloid leukaemia.

Barbarroja N, Arístides-Torres L, Hernandez V, Martín C, Dorado G, Torres A, Velasco F, Lopez-Pedrera C. Barbarroja N, et al. Leuk Lymphoma. 2007 Jun;48(6):1187-99. doi: 10.1080/10428190701340616. Leuk Lymphoma. 2007. PMID: 17577783

9

MEK inhibition induces caspases activation, differentiation blockade and PML/RARalpha degradation in acute promyelocytic leukaemia.

Barbarroja N, Siendones E, Torres LA, Luque MJ, Martinez JM, Dorado G, Velasco F, Torres A, López-Pedrera C. Barbarroja N, et al. Br J Haematol. 2008 Jul;142(1):27-35. doi: 10.1111/j.1365-2141.2008.07154.x. Epub 2008 Apr 28. Br J Haematol. 2008. PMID: 18445086 Free article.

10

[Intracellular mechanisms involved in the angiogenic process in hematological malignancies].

Barbarroja N, Velasco F, López-Pedrera C. Barbarroja N, et al. Med Clin (Barc). 2008 Apr 26;130(15):585-90. doi: 10.1157/13119981. Med Clin (Barc). 2008. PMID: 18462638 Review. Spanish.

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