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Thirty-Eight-Negative Kinase 1 Is a Mediator of Acute Kidney Injury in Experimental and Clinical Traumatic Hemorrhagic Shock.
Halbgebauer R, Karasu E, Braun CK, Palmer A, Braumüller S, Schultze A, Schäfer F, Bückle S, Eigner A, Wachter U, Radermacher P, Resuello RRG, Tuplano JV, Nilsson Ekdahl K, Nilsson B, Armacki M, Kleger A, Seufferlein T, Kalbitz M, Gebhard F, Lambris JD, van Griensven M, Huber-Lang M. Halbgebauer R, et al. Front Immunol. 2020 Aug 26;11:2081. doi: 10.3389/fimmu.2020.02081. eCollection 2020. Front Immunol. 2020. PMID: 32983160 Free PMC article. Clinical Trial.
Generation of C5a in the absence of C3: a new complement activation pathway.
Huber-Lang M, Sarma JV, Zetoune FS, Rittirsch D, Neff TA, McGuire SR, Lambris JD, Warner RL, Flierl MA, Hoesel LM, Gebhard F, Younger JG, Drouin SM, Wetsel RA, Ward PA. Huber-Lang M, et al. Nat Med. 2006 Jun;12(6):682-7. doi: 10.1038/nm1419. Epub 2006 May 21. Nat Med. 2006. PMID: 16715088
Complement activation during sepsis in humans.
Schreiber H, Rittirsch D, Flierl M, Brueckner U, Schneider M, Weiss M, Gebhard F, Huber-Lang M. Schreiber H, et al. Adv Exp Med Biol. 2006;586:217-26. doi: 10.1007/0-387-34134-X_15. Adv Exp Med Biol. 2006. PMID: 16893075 No abstract available.
353 results