White DL - Search Results

1

Lineage of measurable residual disease in patients with chronic myeloid leukemia in treatment-free remission.

Pagani IS, Dang P, Saunders VA, Grose R, Shanmuganathan N, Kok CH, Carne L, Rwodzi Z, Watts S, McLean J, Braley J, Altamura H, Yeung DT, Branford S, Yong ASM, White DL, Hughes TP, Ross DM. Pagani IS, et al. Among authors: white dl. Leukemia. 2020 Apr;34(4):1052-1061. doi: 10.1038/s41375-019-0647-x. Epub 2019 Nov 25. Leukemia. 2020. PMID: 31768016

2

Direct analysis of FACS-sorted hemopoietic cell fractions using FISH.

White DL, Hutchins CJ, Haylock DN, Turczynowicz S, Bishop A, To LB, Hughes TP, Juttner CA. White DL, et al. Biotechniques. 1995 May;18(5):818, 820-1. Biotechniques. 1995. PMID: 7542458 No abstract available.

3

Apoptosis regulatory gene NEDD2 maps to human chromosome segment 7q34-35, a region frequently affected in haematological neoplasms.

Kumar S, White DL, Takai S, Turczynowicz S, Juttner CA, Hughes TP. Kumar S, et al. Among authors: white dl. Hum Genet. 1995 Jun;95(6):641-4. doi: 10.1007/BF00209480. Hum Genet. 1995. PMID: 7789948

4

Detection of minimal residual disease in an AML patient with trisomy 8 using interphase fish.

White DL, Hutchins CJ, Turczynowicz S, Suttle J, Haylock DN, Hughes TP, Juttner CA, To LB. White DL, et al. Pathology. 1997 Aug;29(3):289-93. doi: 10.1080/00313029700169115. Pathology. 1997. PMID: 9271020

5

Successful peripheral blood stem cell mobilisation with filgrastim in patients with chronic myeloid leukaemia achieving complete cytogenetic response with imatinib, without increasing disease burden as measured by quantitative real-time PCR.

Hui CH, Goh KY, White D, Branford S, Grigg A, Seymour JF, Kwan YL, Walsh S, Hoyt R, Trickett A, Rudzki B, Ma DD, To LB, Hughes TP. Hui CH, et al. Leukemia. 2003 May;17(5):821-8. doi: 10.1038/sj.leu.2402917. Leukemia. 2003. PMID: 12750692

6

In vitro sensitivity to imatinib-induced inhibition of ABL kinase activity is predictive of molecular response in patients with de novo CML.

White D, Saunders V, Lyons AB, Branford S, Grigg A, To LB, Hughes T. White D, et al. Blood. 2005 Oct 1;106(7):2520-6. doi: 10.1182/blood-2005-03-1103. Epub 2005 Jun 14. Blood. 2005. PMID: 15956284 Free article.

7

OCT-1-mediated influx is a key determinant of the intracellular uptake of imatinib but not nilotinib (AMN107): reduced OCT-1 activity is the cause of low in vitro sensitivity to imatinib.

White DL, Saunders VA, Dang P, Engler J, Zannettino AC, Cambareri AC, Quinn SR, Manley PW, Hughes TP. White DL, et al. Blood. 2006 Jul 15;108(2):697-704. doi: 10.1182/blood-2005-11-4687. Epub 2006 Apr 4. Blood. 2006. PMID: 16597591 Free article.

8

Imatinib increases the intracellular concentration of nilotinib, which may explain the observed synergy between these drugs.

White DL, Saunders VA, Quinn SR, Manley PW, Hughes TP. White DL, et al. Blood. 2007 Apr 15;109(8):3609-10. doi: 10.1182/blood-2006-11-058032. Blood. 2007. PMID: 17409347 Free article. No abstract available.

9

Most CML patients who have a suboptimal response to imatinib have low OCT-1 activity: higher doses of imatinib may overcome the negative impact of low OCT-1 activity.

White DL, Saunders VA, Dang P, Engler J, Venables A, Zrim S, Zannettino A, Lynch K, Manley PW, Hughes T. White DL, et al. Blood. 2007 Dec 1;110(12):4064-72. doi: 10.1182/blood-2007-06-093617. Epub 2007 Aug 30. Blood. 2007. PMID: 17761829 Free article. Clinical Trial.

10

Measurement of in vivo BCR-ABL kinase inhibition to monitor imatinib-induced target blockade and predict response in chronic myeloid leukemia.

White D, Saunders V, Grigg A, Arthur C, Filshie R, Leahy MF, Lynch K, To LB, Hughes T. White D, et al. J Clin Oncol. 2007 Oct 1;25(28):4445-51. doi: 10.1200/JCO.2006.09.9499. J Clin Oncol. 2007. PMID: 17906206

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