Müller UC - Search Results

1

Acute function of secreted amyloid precursor protein fragment APPsα in synaptic plasticity.

Hick M, Herrmann U, Weyer SW, Mallm JP, Tschäpe JA, Borgers M, Mercken M, Roth FC, Draguhn A, Slomianka L, Wolfer DP, Korte M, Müller UC. Hick M, et al. Among authors: muller uc. Acta Neuropathol. 2015 Jan;129(1):21-37. doi: 10.1007/s00401-014-1368-x. Epub 2014 Nov 29. Acta Neuropathol. 2015. PMID: 25432317

2

Behavioral and anatomical deficits in mice homozygous for a modified beta-amyloid precursor protein gene.

Müller U, Cristina N, Li ZW, Wolfer DP, Lipp HP, Rülicke T, Brandner S, Aguzzi A, Weissmann C. Müller U, et al. Cell. 1994 Dec 2;79(5):755-65. doi: 10.1016/0092-8674(94)90066-3. Cell. 1994. PMID: 8001115

3

Mice homozygous for a modified beta-amyloid precursor protein (beta APP) gene show impaired behavior and high incidence of agenesis of the corpus callosum.

Müller U, Cristina N, Li ZW, Wolfer DP, Lipp HP, Rülicke T, Brandner S, Aguzzi A, Weissman C. Müller U, et al. Ann N Y Acad Sci. 1996 Jan 17;777:65-73. doi: 10.1111/j.1749-6632.1996.tb34402.x. Ann N Y Acad Sci. 1996. PMID: 8624128

4

Assessing the effects of the 129/Sv genetic background on swimming navigation learning in transgenic mutants: a study using mice with a modified beta-amyloid precursor protein gene.

Wolfer DP, Müller U, Stagliar M, Lipp HP. Wolfer DP, et al. Brain Res. 1997 Oct 10;771(1):1-13. doi: 10.1016/s0006-8993(97)00673-2. Brain Res. 1997. PMID: 9383002

5

Neurobehavioral development, adult openfield exploration and swimming navigation learning in mice with a modified beta-amyloid precursor protein gene.

Tremml P, Lipp HP, Müller U, Ricceri L, Wolfer DP. Tremml P, et al. Behav Brain Res. 1998 Sep;95(1):65-76. doi: 10.1016/s0166-4328(97)00211-8. Behav Brain Res. 1998. PMID: 9754878

6

Genetic background changes the pattern of forebrain commissure defects in transgenic mice underexpressing the beta-amyloid-precursor protein.

Magara F, Müller U, Li ZW, Lipp HP, Weissmann C, Stagljar M, Wolfer DP. Magara F, et al. Proc Natl Acad Sci U S A. 1999 Apr 13;96(8):4656-61. doi: 10.1073/pnas.96.8.4656. Proc Natl Acad Sci U S A. 1999. PMID: 10200318 Free PMC article.

7

Mice with combined gene knock-outs reveal essential and partially redundant functions of amyloid precursor protein family members.

Heber S, Herms J, Gajic V, Hainfellner J, Aguzzi A, Rülicke T, von Kretzschmar H, von Koch C, Sisodia S, Tremml P, Lipp HP, Wolfer DP, Müller U. Heber S, et al. J Neurosci. 2000 Nov 1;20(21):7951-63. doi: 10.1523/JNEUROSCI.20-21-07951.2000. J Neurosci. 2000. PMID: 11050115 Free PMC article.

8

Enriched early experiences of mice underexpressing the beta-amyloid precursor protein restore spatial learning capabilities but not normal openfield behavior of adult animals.

Tremml P, Lipp HP, Müller U, Wolfer DP. Tremml P, et al. Genes Brain Behav. 2002 Nov;1(4):230-41. doi: 10.1034/j.1601-183x.2002.10405.x. Genes Brain Behav. 2002. PMID: 12882368

9

The secreted beta-amyloid precursor protein ectodomain APPs alpha is sufficient to rescue the anatomical, behavioral, and electrophysiological abnormalities of APP-deficient mice.

Ring S, Weyer SW, Kilian SB, Waldron E, Pietrzik CU, Filippov MA, Herms J, Buchholz C, Eckman CB, Korte M, Wolfer DP, Müller UC. Ring S, et al. Among authors: muller uc. J Neurosci. 2007 Jul 18;27(29):7817-26. doi: 10.1523/JNEUROSCI.1026-07.2007. J Neurosci. 2007. PMID: 17634375 Free PMC article.

10

Embryonic stem cell-derived neurons as a cellular system to study gene function: lack of amyloid precursor proteins APP and APLP2 leads to defective synaptic transmission.

Schrenk-Siemens K, Perez-Alcala S, Richter J, Lacroix E, Rahuel J, Korte M, Müller U, Barde YA, Bibel M. Schrenk-Siemens K, et al. Stem Cells. 2008 Aug;26(8):2153-63. doi: 10.1634/stemcells.2008-0010. Epub 2008 Jun 5. Stem Cells. 2008. PMID: 18535156

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