Okazawa H - Search Results

1

In utero gene therapy rescues microcephaly caused by Pqbp1-hypofunction in neural stem progenitor cells.

Ito H, Shiwaku H, Yoshida C, Homma H, Luo H, Chen X, Fujita K, Musante L, Fischer U, Frints SG, Romano C, Ikeuchi Y, Shimamura T, Imoto S, Miyano S, Muramatsu SI, Kawauchi T, Hoshino M, Sudol M, Arumughan A, Wanker EE, Rich T, Schwartz C, Matsuzaki F, Bonni A, Kalscheuer VM, Okazawa H. Ito H, et al. Among authors: okazawa h. Mol Psychiatry. 2015 Apr;20(4):459-71. doi: 10.1038/mp.2014.69. Epub 2014 Jul 29. Mol Psychiatry. 2015. PMID: 25070536 Free PMC article.

2

PQBP-1 (Np/PQ): a polyglutamine tract-binding and nuclear inclusion-forming protein.

Okazawa H, Sudol M, Rich T. Okazawa H, et al. Brain Res Bull. 2001 Oct-Nov 1;56(3-4):273-80. doi: 10.1016/s0361-9230(01)00579-2. Brain Res Bull. 2001. PMID: 11719261 Review.

3

Interaction between mutant ataxin-1 and PQBP-1 affects transcription and cell death.

Okazawa H, Rich T, Chang A, Lin X, Waragai M, Kajikawa M, Enokido Y, Komuro A, Kato S, Shibata M, Hatanaka H, Mouradian MM, Sudol M, Kanazawa I. Okazawa H, et al. Neuron. 2002 May 30;34(5):701-13. doi: 10.1016/s0896-6273(02)00697-9. Neuron. 2002. PMID: 12062018 Free article.

4

Mutant huntingtin promotes the fibrillogenesis of wild-type huntingtin: a potential mechanism for loss of huntingtin function in Huntington's disease.

Busch A, Engemann S, Lurz R, Okazawa H, Lehrach H, Wanker EE. Busch A, et al. Among authors: okazawa h. J Biol Chem. 2003 Oct 17;278(42):41452-61. doi: 10.1074/jbc.M303354200. Epub 2003 Jul 29. J Biol Chem. 2003. PMID: 12888569 Free article.

5

Histone deacetylase activity is retained in primary neurons expressing mutant huntingtin protein.

Hoshino M, Tagawa K, Okuda T, Murata M, Oyanagi K, Arai N, Mizutani T, Kanazawa I, Wanker EE, Okazawa H. Hoshino M, et al. Among authors: okazawa h. J Neurochem. 2003 Oct;87(1):257-67. doi: 10.1046/j.1471-4159.2003.01991.x. J Neurochem. 2003. PMID: 12969272 Free article.

6

General transcriptional repression by polyglutamine disease proteins is not directly linked to the presence of inclusion bodies.

Hoshino M, Tagawa K, Okuda T, Okazawa H. Hoshino M, et al. Among authors: okazawa h. Biochem Biophys Res Commun. 2004 Jan 2;313(1):110-6. doi: 10.1016/j.bbrc.2003.11.086. Biochem Biophys Res Commun. 2004. PMID: 14672705

7

Distinct aggregation and cell death patterns among different types of primary neurons induced by mutant huntingtin protein.

Tagawa K, Hoshino M, Okuda T, Ueda H, Hayashi H, Engemann S, Okado H, Ichikawa M, Wanker EE, Okazawa H. Tagawa K, et al. Among authors: okazawa h. J Neurochem. 2004 May;89(4):974-87. doi: 10.1111/j.1471-4159.2004.02372.x. J Neurochem. 2004. PMID: 15140196 Free article.

8

Oct-3/4 repression accelerates differentiation of neural progenitor cells in vitro and in vivo.

Okuda T, Tagawa K, Qi ML, Hoshio M, Ueda H, Kawano H, Kanazawa I, Muramatsu M, Okazawa H. Okuda T, et al. Among authors: okazawa h. Brain Res Mol Brain Res. 2004 Dec 6;132(1):18-30. doi: 10.1016/j.molbrainres.2004.08.021. Brain Res Mol Brain Res. 2004. PMID: 15548425

9

Polyglutamine tract-binding protein-1 dysfunction induces cell death of neurons through mitochondrial stress.

Marubuchi S, Wada Y, Okuda T, Hara Y, Qi ML, Hoshino M, Nakagawa M, Kanazawa I, Okazawa H. Marubuchi S, et al. Among authors: okazawa h. J Neurochem. 2005 Nov;95(3):858-70. doi: 10.1111/j.1471-4159.2005.03405.x. Epub 2005 Aug 16. J Neurochem. 2005. PMID: 16104847 Free article.

10

PQBP-1 is expressed predominantly in the central nervous system during development.

Qi Y, Hoshino M, Wada Y, Marubuchi S, Yoshimura N, Kanazawa I, Shinomiya K, Okazawa H. Qi Y, et al. Among authors: okazawa h. Eur J Neurosci. 2005 Sep;22(6):1277-86. doi: 10.1111/j.1460-9568.2005.04339.x. Eur J Neurosci. 2005. PMID: 16190883

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