Baes M - Search Results

1

Generation of Pex5-loxP mice allowing the conditional elimination of peroxisomes.

Baes M, Dewerchin M, Janssen A, Collen D, Carmeliet P. Baes M, et al. Genesis. 2002 Feb;32(2):177-8. doi: 10.1002/gene.10047. Genesis. 2002. PMID: 11857813 No abstract available.

2

The glucocorticoid hormone dexamethasone reverses the growth hormone-releasing properties of the cholinomimetic carbachol.

Carmeliet P, Baes M, Denef C. Carmeliet P, et al. Among authors: baes m. Endocrinology. 1989 May;124(5):2625-34. doi: 10.1210/endo-124-5-2625. Endocrinology. 1989. PMID: 2707169

3

Sequence requirements for high affinity retinoid X receptor-alpha homodimer binding.

Castelein H, Janssen A, Declercq PE, Baes M. Castelein H, et al. Among authors: baes m. Mol Cell Endocrinol. 1996 May 17;119(1):11-20. doi: 10.1016/0303-7207(96)03794-x. Mol Cell Endocrinol. 1996. PMID: 8793849

4

A mouse model for Zellweger syndrome.

Baes M, Gressens P, Baumgart E, Carmeliet P, Casteels M, Fransen M, Evrard P, Fahimi D, Declercq PE, Collen D, van Veldhoven PP, Mannaerts GP. Baes M, et al. Nat Genet. 1997 Sep;17(1):49-57. doi: 10.1038/ng0997-49. Nat Genet. 1997. PMID: 9288097

5

Urokinase-generated plasmin activates matrix metalloproteinases during aneurysm formation.

Carmeliet P, Moons L, Lijnen R, Baes M, Lemaître V, Tipping P, Drew A, Eeckhout Y, Shapiro S, Lupu F, Collen D. Carmeliet P, et al. Among authors: baes m. Nat Genet. 1997 Dec;17(4):439-44. doi: 10.1038/ng1297-439. Nat Genet. 1997. PMID: 9398846

6

Inhibition of plasminogen activators or matrix metalloproteinases prevents cardiac rupture but impairs therapeutic angiogenesis and causes cardiac failure.

Heymans S, Luttun A, Nuyens D, Theilmeier G, Creemers E, Moons L, Dyspersin GD, Cleutjens JP, Shipley M, Angellilo A, Levi M, Nübe O, Baker A, Keshet E, Lupu F, Herbert JM, Smits JF, Shapiro SD, Baes M, Borgers M, Collen D, Daemen MJ, Carmeliet P. Heymans S, et al. Among authors: baes m. Nat Med. 1999 Oct;5(10):1135-42. doi: 10.1038/13459. Nat Med. 1999. PMID: 10502816

7

Docosahexaenoic acid deficit is not a major pathogenic factor in peroxisome-deficient mice.

Janssen A, Baes M, Gressens P, Mannaerts GP, Declercq P, Van Veldhoven PP. Janssen A, et al. Among authors: baes m. Lab Invest. 2000 Jan;80(1):31-5. doi: 10.1038/labinvest.3780005. Lab Invest. 2000. PMID: 10653000 Free article.

8

Inactivation of the peroxisomal multifunctional protein-2 in mice impedes the degradation of not only 2-methyl-branched fatty acids and bile acid intermediates but also of very long chain fatty acids.

Baes M, Huyghe S, Carmeliet P, Declercq PE, Collen D, Mannaerts GP, Van Veldhoven PP. Baes M, et al. J Biol Chem. 2000 May 26;275(21):16329-36. doi: 10.1074/jbc.M001994200. J Biol Chem. 2000. PMID: 10748062 Free article.

9

Prenatal and postnatal development of peroxisomal lipid-metabolizing pathways in the mouse.

Huyghe S, Casteels M, Janssen A, Meulders L, Mannaerts GP, Declercq PE, Van Veldhoven PP, Baes M. Huyghe S, et al. Among authors: baes m. Biochem J. 2001 Feb 1;353(Pt 3):673-80. doi: 10.1042/0264-6021:3530673. Biochem J. 2001. PMID: 11171065 Free PMC article.

10

The neuronal migration defect in mice with Zellweger syndrome (Pex5 knockout) is not caused by the inactivity of peroxisomal beta-oxidation.

Baes M, Gressens P, Huyghe S, De NK, Qi C, Jia Y, Mannaerts GP, Evrard P, Van VP, Declercq PE, Reddy JK. Baes M, et al. J Neuropathol Exp Neurol. 2002 Apr;61(4):368-74. doi: 10.1093/jnen/61.4.368. J Neuropathol Exp Neurol. 2002. PMID: 11939592

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