Activated protein C resistance and the factor V Leiden mutation in children with thrombosis

Am J Hematol. 1998 Jan;57(1):29-32. doi: 10.1002/(sici)1096-8652(199801)57:1<29::aid-ajh5>3.0.co;2-3.

Abstract

To determine the prevalence of activated protein C resistance and the factor V Leiden mutation (position 1691, arginine 506 to glutamine substitution) in children with thrombosis, plasma samples from children with thrombosis were tested for activated protein C resistance. DNA was analyzed for the factor V Leiden mutation. Five of 34 children (15%) had activated protein C resistance; each was heterozygous for the factor V Leiden mutation. All 5 children heterozygous for the factor V Leiden mutation suffered non-CNS venous thromboses comprising 21% of the group of children (5/24) with non-CNS venous thrombotic events. Each of these 5 children had a family history of thrombosis. In conclusion, children with non-CNS venous thrombosis should be evaluated for the factor V Leiden mutation. Children most likely affected are those with a family history of thrombosis.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adolescent
  • Antithrombin III Deficiency
  • Blood Coagulation
  • Child
  • Factor V / analysis*
  • Female
  • Humans
  • Male
  • Partial Thromboplastin Time
  • Point Mutation
  • Protein C / genetics*
  • Thrombosis / genetics*

Substances

  • Protein C
  • factor V Leiden
  • Factor V