Genetic testing for familial cancer. Consequences of RET proto-oncogene mutation analysis in multiple endocrine neoplasia, type 2

Arch Surg. 1997 Sep;132(9):1022-5. doi: 10.1001/archsurg.1997.01430330088015.

Abstract

Objective: To assess clinician use and acceptance of RET proto-oncogene mutation testing in multiple endocrine neoplasia, type 2 (MEN 2) family members.

Design: A retrospective survey of clinicians managing 26 MEN 2 families with documented RET mutations to assess the effect of genetic screening on subsequent investigation and management of family members.

Setting: Tertiary referral center for RET mutation testing.

Main outcome measures: The screening procedures used by clinicians and the altered incidence of C-cell hyperplasia vs medullary thyroid carcinoma in genetically as opposed to biochemically identified affected family members.

Results: Among RET mutation-positive patients, thyroidectomy performed for clinical or biochemical indication disclosed medullary thyroid carcinoma in 44 (98%) of 45 patients and precursor C-cell hyperplasia in only 1 (2%) patient. When prophylactic thyroidectomy was performed based on a positive genetic result, medullary thyroid carcinoma occurred in 3 (43%) of 7 patients and C-cell hyperplasia in 4 (57%) of 7 patients (P < .001). RET mutation-negative patients were not subjected to further biochemical testing, but 4 had already undergone thyroidectomy based on abnormal results of pentagastrin stimulation tests, including 2 patients who were known to be RET mutation-negative at the time of surgery. RET mutation testing was well accepted and resulted in additional family members consenting to screening in more than 85% of families.

Conclusions: Genetic screening for RET proto-oncogene mutations in MEN 2 is a powerful diagnostic tool that enables prophylactic thyroidectomy to be performed in RET mutation-positive patients at an earlier stage of the disease process than does traditional biochemical screening.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Aged, 80 and over
  • Animals
  • Carcinoma, Medullary / genetics*
  • Carcinoma, Medullary / pathology
  • Child
  • Child, Preschool
  • Female
  • Genetic Testing / methods*
  • Germ-Line Mutation / genetics*
  • Guinea Pigs
  • Humans
  • Male
  • Middle Aged
  • Multiple Endocrine Neoplasia Type 2a / genetics*
  • Multiple Endocrine Neoplasia Type 2a / pathology
  • Multiple Endocrine Neoplasia Type 2b / genetics*
  • Multiple Endocrine Neoplasia Type 2b / pathology
  • Proto-Oncogene Mas
  • Proto-Oncogenes / genetics*
  • Receptor Protein-Tyrosine Kinases / genetics*
  • Retrospective Studies
  • Thyroid Gland / pathology
  • Thyroid Neoplasms / genetics*
  • Thyroid Neoplasms / pathology
  • Thyroidectomy

Substances

  • MAS1 protein, human
  • Proto-Oncogene Mas
  • Receptor Protein-Tyrosine Kinases