Genome-wide hypermutation in a subpopulation of stationary-phase cells underlies recombination-dependent adaptive mutation

EMBO J. 1997 Jun 2;16(11):3303-11. doi: 10.1093/emboj/16.11.3303.

Abstract

Stationary-phase mutation in microbes can produce selected ('adaptive') mutants preferentially. In one system, this occurs via a distinct, recombination-dependent mechanism. Two points of controversy have surrounded these adaptive reversions of an Escherichia coli lac mutation. First, are the mutations directed preferentially to the selected gene in a Lamarckian manner? Second, is the adaptive mutation mechanism specific to the F plasmid replicon carrying lac? We report that lac adaptive mutations are associated with hypermutation in unselected genes, in all replicons in the cell. The associated mutations have a similar sequence spectrum to the adaptive reversions. Thus, the adaptive mutagenesis mechanism is not directed to the lac genes, in a Lamarckian manner, nor to the F' replicon carrying lac. Hypermutation was not found in non-revertants exposed to selection. Therefore, the genome-wide hypermutation underlying adaptive mutation occurs in a differentiated subpopulation. The existence of mutable subpopulations in non-growing cells is important in bacterial evolution and could be relevant to the somatic mutations that give rise to cancers in multicellular organisms.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adaptation, Biological / genetics*
  • Base Sequence
  • Biological Evolution*
  • Chromosomes, Bacterial
  • Escherichia coli / genetics*
  • Escherichia coli / growth & development
  • F Factor / genetics
  • Genome, Bacterial*
  • Lac Operon / genetics
  • Models, Genetic
  • Molecular Sequence Data
  • Mutagenesis*
  • Recombination, Genetic*
  • Selection, Genetic
  • Time Factors