Evidence of a hypercoagulable state in patients with acute lymphoblastic leukemia treated with low dose of E. coli L-asparaginase: a GIMEMA study

Thromb Haemost. 1993 Jan 11;69(1):12-5.

Abstract

Blood coagulation abnormalities induced by administration of E. coli L-asparaginase were investigated in 25 patients with acute lymphoblastic leukemia treated according to the GIMEMA ALL 0288 trial. Dosage of L-asparaginase was relatively low (6,000 U/m2/day for 7 days total dose 42,000 U/m2) as compared to the conventional dosages (120,000-140,000 U/m2 over 10-14 days). A significant decrease in fibronogen, plasminogen, alpha2-antiplasmin and antithrombin III was observed from day IV of L-asparaginase and it was maximum on day VIII, with return to the baseline levels on day XV. Protein C levels had only a borderline reduction, while no modification of protein S or factor VII was observed. Two of the patients investigated developed thrombosis. The presence of a prothrombotic state induced even by this low dosage of E. coli L-asparaginase was suggested by a significant increase of sensitive markers of hypercoagulability such as fibrinopeptide A, thrombin-antithrombin complexes, and prothrombin fragment F1 + 2.

MeSH terms

  • Adolescent
  • Adult
  • Asparaginase / administration & dosage
  • Asparaginase / adverse effects*
  • Blood Coagulation Disorders / chemically induced*
  • Blood Coagulation Factors / metabolism
  • Escherichia coli / enzymology*
  • Female
  • Humans
  • Incidence
  • Male
  • Middle Aged
  • Precursor Cell Lymphoblastic Leukemia-Lymphoma / blood
  • Precursor Cell Lymphoblastic Leukemia-Lymphoma / drug therapy*
  • Sensitivity and Specificity
  • Thrombin / biosynthesis*
  • Thrombophlebitis / chemically induced
  • Thrombophlebitis / epidemiology

Substances

  • Blood Coagulation Factors
  • Thrombin
  • Asparaginase