Expression of c-myc in progenitor cells of the bronchopulmonary epithelium and in a large number of non-small cell lung cancers

Am J Respir Cell Mol Biol. 1993 Jul;9(1):33-43. doi: 10.1165/ajrcmb/9.1.33.

Abstract

We performed in situ hybridization for c-myc, N-myc, and L-myc mRNA expression using 35S-labeled cRNA probes on frozen sections of 19 pairs of non-small cell lung cancers (NSCLC) and the surrounding non-neoplastic lung tissue. In non-neoplastic lung, c-myc expression was strongest in bronchial epithelium basal cells and hyperplastic alveolar type II pneumocytes, which are potential progenitor cells for bronchopulmonary epithelium and their tumors. In contrast, N-myc and L-myc mRNAs were not detected in non-neoplastic lung. In studies of freshly resected primary tumors, expression of c-myc was detected in 11 of 19 NSCLC (with the highest levels in squamous cell carcinomas), two of which also expressed L-myc, while N-myc expression was never detected. Levels of c-myc expression in tumors were significantly higher than in non-neoplastic lung samples. We conclude that: (1) c-myc expression in non-neoplastic lung tissues is highest in bronchial basal cells and hyperplastic type II cells, and (2) in NSCLC, overexpression of the myc-proto-oncogene is common.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Blotting, Northern
  • Bronchi / cytology
  • Bronchi / metabolism*
  • Carcinoma, Non-Small-Cell Lung / metabolism*
  • Epithelial Cells
  • Epithelium / metabolism
  • Gene Expression
  • Genes, myc*
  • Humans
  • Immunohistochemistry
  • In Situ Hybridization
  • Ki-67 Antigen
  • Lung Neoplasms / metabolism*
  • Multigene Family
  • Neoplasm Proteins / biosynthesis
  • Nuclear Proteins / biosynthesis
  • Proto-Oncogene Mas
  • Stem Cells / metabolism*

Substances

  • Ki-67 Antigen
  • MAS1 protein, human
  • Neoplasm Proteins
  • Nuclear Proteins
  • Proto-Oncogene Mas