Interaction of mesangial cells with IgA and IgG immune complexes: a possible mechanism of glomerular injury in IgA nephropathy

Contrib Nephrol. 1993:104:127-37. doi: 10.1159/000422405.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antigen-Antibody Complex / metabolism*
  • Dinoprostone / biosynthesis
  • Glomerular Mesangium / metabolism*
  • Glomerular Mesangium / pathology
  • Glomerulonephritis, IGA / etiology
  • Glomerulonephritis, IGA / immunology*
  • Glomerulonephritis, IGA / pathology
  • Humans
  • Immunoglobulin A / metabolism*
  • Immunoglobulin G / metabolism*
  • Inflammation
  • Myeloma Proteins / metabolism
  • Platelet Activating Factor / biosynthesis
  • Receptors, Fc*
  • Receptors, Immunologic / metabolism
  • Superoxides / metabolism
  • Tumor Necrosis Factor-alpha / biosynthesis

Substances

  • Antigen-Antibody Complex
  • IgA receptor
  • Immunoglobulin A
  • Immunoglobulin G
  • Myeloma Proteins
  • Platelet Activating Factor
  • Receptors, Fc
  • Receptors, Immunologic
  • Tumor Necrosis Factor-alpha
  • Superoxides
  • Dinoprostone