Abstract
We report on four patients with the Smith-Lemli-Opitz (SLO) syndrome who appear to have a defect in cholesterol biosynthesis. The initial results of therapy of one of the patients with cholesterol and bile acids to correct her metabolic abnormalities are described. This finding provides a biochemical marker to help in the diagnosis of this syndrome, may provide insight into the pathogenesis of this disorder, and have therapeutic and prenatal diagnostic implications as well.
MeSH terms
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Abnormalities, Multiple / diet therapy
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Abnormalities, Multiple / metabolism*
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Adolescent
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Bile Acids and Salts / biosynthesis
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Child
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Child, Preschool
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Cholesterol / biosynthesis*
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Cholesterol, Dietary / therapeutic use
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Dehydrocholesterols / blood
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Face / abnormalities
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Female
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Genes, Recessive
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Humans
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Infant
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Intellectual Disability* / diet therapy
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Intellectual Disability* / metabolism
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Lipid Metabolism, Inborn Errors / diet therapy
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Lipid Metabolism, Inborn Errors / metabolism*
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Male
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Microcephaly
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Oxidoreductases / deficiency
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Oxidoreductases Acting on CH-CH Group Donors*
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Sterols / blood
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Syndrome
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Ursodeoxycholic Acid / therapeutic use
Substances
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Bile Acids and Salts
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Cholesterol, Dietary
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Dehydrocholesterols
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Sterols
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Ursodeoxycholic Acid
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Cholesterol
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7-dehydrocholesterol
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Oxidoreductases
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Oxidoreductases Acting on CH-CH Group Donors
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7-dehydrocholesterol reductase