Superantigen-reactive human T cells express a biased repertoire of T-cell receptor V beta joining regions

Res Immunol. 1994 Sep;145(7):517-31. doi: 10.1016/s0923-2494(94)80070-7.

Abstract

A major characteristic of superantigens is their ability to stimulate T cells based predominantly on the type of variable segment of the T-cell receptor (TCR) V beta chain. Recently, however, reports from several laboratories have also implied a role for non-V beta elements in superantigen binding. The goal of the present study was to determine whether TCR V beta-D beta-J beta joining sequences may influence the interaction of superantigens with their target cells. To ascertain how the actual TCR repertoire of superantigen-triggered cells deviates from the theoretical one, we generated a large panel of joining region sequences from TCR carrying the TCR V beta 12 and TCR V beta 5,1 regions. The 245 sequences analysed represent transcripts of T cells from the same donor triggered either with an anti-CD3 monoclonal antibody or with the Staphylococcus aureus enterotoxins. Comparison of the joining sequences of these different groups demonstrates a skewed J beta usage in the sequences derived from superantigen-triggered cells and also provides evidence that ascribes to the putative CDR3 region of V beta segments a role in superantigen recognition. Finally, the data presented give some hints of the regions of the putative CDR3 loop that may play a major role in this function.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Antibodies, Monoclonal / immunology
  • Base Sequence
  • DNA Nucleotidyltransferases / metabolism
  • DNA, Complementary / genetics
  • Gene Rearrangement, beta-Chain T-Cell Antigen Receptor*
  • Humans
  • Lymphocyte Activation
  • Molecular Sequence Data
  • Polymerase Chain Reaction
  • RNA, Complementary / genetics
  • Receptors, Antigen, T-Cell, alpha-beta / genetics*
  • Sequence Alignment
  • Superantigens / immunology*
  • T-Lymphocyte Subsets / immunology*
  • VDJ Recombinases

Substances

  • Antibodies, Monoclonal
  • DNA, Complementary
  • RNA, Complementary
  • Receptors, Antigen, T-Cell, alpha-beta
  • Superantigens
  • DNA Nucleotidyltransferases
  • VDJ Recombinases