Hepatitis B virus unable to secrete e antigen and response to interferon in chronic hepatitis B

Gastroenterology. 1993 Sep;105(3):845-50. doi: 10.1016/0016-5085(93)90903-p.

Abstract

Background: Anti-hepatitis e antigen-positive chronic hepatitis B is a progressive liver disease associated with precore mutant hepatitis B virus (HBV) and poor response to interferon. Therefore, precore mutant HBV may behave as an interferon-resistant virus. The relations between the prevalences of wild-type and precore mutant HBVs in baseline viremias and response to interferon were analyzed.

Methods: Sera from 115 patients (59 treated and 56 untreated, followed up for 30 months) were tested using a quantitative oligonucleotide hybridization assay.

Results: Spontaneous or interferon-induced recoveries were observed in 28.5% (6 of 21) and 47.3% (18 of 38) or in 0% (0 of 35) and 19% (4 of 21) of the patients with wild-type prevalent or mutant prevalent HBVs, respectively. Relapses occurred in 85.7% (12 of 14) and 19.4% (4 of 21) of treated patients with prevalent precore mutant and prevalent wild-type HBV, respectively (P = 0.0001). High precore mutant HBV levels (> 20% of total viremia) were associated with the lack of permanent response to interferon (P = 0.01).

Conclusions: Precore mutant HBV can influence the response to interferon when it reaches significant serum levels (> 20% of total viremia). Therefore, chronic hepatitis B should be treated as early as possible in its natural history before precore mutant HBV is selected as a prevalent virus.

Publication types

  • Clinical Trial
  • Randomized Controlled Trial

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Analysis of Variance
  • Base Sequence
  • DNA, Viral / analysis
  • DNA, Viral / genetics
  • Female
  • Hepatitis B / drug therapy
  • Hepatitis B / immunology*
  • Hepatitis B / pathology
  • Hepatitis B e Antigens / analysis
  • Hepatitis B e Antigens / metabolism*
  • Hepatitis B virus / genetics
  • Hepatitis B virus / isolation & purification
  • Hepatitis B virus / metabolism*
  • Humans
  • Immunohistochemistry
  • Interferons / pharmacology*
  • Interferons / therapeutic use
  • Liver / metabolism
  • Liver / pathology
  • Male
  • Middle Aged
  • Molecular Sequence Data
  • Mutation / genetics
  • Nucleic Acid Hybridization
  • Oligonucleotides
  • Viral Core Proteins / genetics

Substances

  • DNA, Viral
  • Hepatitis B e Antigens
  • Oligonucleotides
  • Viral Core Proteins
  • Interferons