Increased DT-diaphorase activity in transformed and tumorigenic pancreatic acinar cells

Cancer Lett. 1995 Sep 4;96(1):9-14. doi: 10.1016/0304-3835(95)03911-f.

Abstract

Pancreatic acinar cells from rats treated in vitro with 5-azacytidine and/or transfected with an activated c-H-ras demonstrated transformation and tumorigenic phenotypes. DT-diaphorase (NAD(P)H:quinone oxidoreductase) activity was determined in these non-tumorigenic (3AP) and tumorigenic cells (T3AP and T5AM). T5AM cells were those treated with 5-azacytidine and further treated with N'-methyl-N'-nitro-nitrosoguanidine. Higher levels of enzyme activity were found in transformed cells when compared to that in control cells (> 15-fold, 3AP cells; > 40-fold, T3AP cells; > 20-fold T5AM cells). In contrast, NADPH-cytochrome c reductase activity was decreased in transformed cells (> 10-fold, 3AP cells; > 20-fold, T3AP cells; > 10-fold, T5AM cells). These studies demonstrated that pancreatic acinar cells are capable of undergoing alterations in enzyme activity patterns when transformed and that DT-diaphorase may be a good marker for malignant transformation.

MeSH terms

  • Animals
  • Antimetabolites, Antineoplastic / pharmacology
  • Azacitidine / pharmacology
  • Cell Transformation, Neoplastic / metabolism*
  • Cells, Cultured
  • Genes, ras
  • In Vitro Techniques
  • Methylation
  • NAD(P)H Dehydrogenase (Quinone) / metabolism*
  • NADH Dehydrogenase / metabolism*
  • Pancreatic Neoplasms / metabolism*
  • Rats
  • Rats, Inbred F344

Substances

  • Antimetabolites, Antineoplastic
  • NAD(P)H Dehydrogenase (Quinone)
  • NADH Dehydrogenase
  • Azacitidine