Interplay of Cellular Nrf2/NF-κB Signalling after Plasma Stimulation of Malignant vs. Non-Malignant Dermal Cells

Int J Mol Sci. 2024 Oct 11;25(20):10967. doi: 10.3390/ijms252010967.

Abstract

Skin cancer is one of the most common malignancies worldwide. Cold atmospheric pressure Plasma (CAP) is increasingly successful in skin cancer therapy, but further research is needed to understand its selective effects on cancer cells at the molecular level. In this study, A431 (squamous cell carcinoma) and HaCaT (non-malignant) cells cultured under identical conditions revealed similar ROS levels but significantly higher antioxidant levels in unstimulated A431 cells, indicating a higher metabolic turnover typical of tumour cells. HaCaT cells, in contrast, showed increased antioxidant levels upon CAP stimulation, reflecting a robust redox adaptation. Specifically, proteins involved in antioxidant pathways, including NF-κB, IκBα, Nrf2, Keap1, IKK, and pIKK, were quantified, and their translocation level upon stimulation was evaluated. CAP treatment significantly elevated Nrf2 nuclear translocation in non-malignant HaCaT cells, indicating a strong protection against oxidative stress, while selectively inducing NF-κB activation in A431 cells, potentially leading to apoptosis. The expression of pro-inflammatory genes like IL-1B, IL-6, and CXCL8 was downregulated in A431 cells upon CAP treatment. Notably, CAP enhanced the expression of antioxidant response genes HMOX1 and GPX1 in non-malignant cells. The differential response between HaCaT and A431 cells underscores the varied antioxidative capacities, contributing to their distinct molecular responses to CAP-induced oxidative stress.

Keywords: NF-κB; Nrf2; cold atmospheric plasma; dermal cells; oxidative stress; translocation.

MeSH terms

  • Antioxidants / metabolism
  • Antioxidants / pharmacology
  • Apoptosis
  • Cell Line, Tumor
  • Glutathione Peroxidase GPX1
  • HaCaT Cells
  • Heme Oxygenase-1 / genetics
  • Heme Oxygenase-1 / metabolism
  • Humans
  • Keratinocytes / metabolism
  • NF-E2-Related Factor 2* / metabolism
  • NF-kappa B* / metabolism
  • Oxidative Stress*
  • Plasma Gases / pharmacology
  • Reactive Oxygen Species / metabolism
  • Signal Transduction*
  • Skin Neoplasms / metabolism
  • Skin Neoplasms / pathology

Substances

  • NF-E2-Related Factor 2
  • NF-kappa B
  • NFE2L2 protein, human
  • Plasma Gases
  • Reactive Oxygen Species
  • Antioxidants
  • Heme Oxygenase-1
  • Glutathione Peroxidase GPX1