A Rare Case of TP63-Associated Lymphopenia Revealed by Newborn Screening Using TREC

Int J Mol Sci. 2024 Oct 9;25(19):10844. doi: 10.3390/ijms251910844.

Abstract

The expanded newborn screening (NBS) program in the Russian Federation was initiated in 2023, among which severe combined immunodeficiency (SCID) is screened using TREC/KREC assays. Here, we report a rare case of a TP63-associated disease identified through this NBS program. Dried blood spots from newborns were initially screened for TREC/KREC levels, and those with values below the cut-off underwent confirmatory testing and further genetic analysis, including whole-exome sequencing (WES). A male newborn was identified with significantly reduced TREC values, indicative of T cell lymphopenia. Genetic analysis revealed a heterozygous NM_003722.5:c.1027C>T variant in TP63, leading to the p.(Arg343Trp) substitution within the DNA binding domain. This mutation has been previously associated with Ectrodactyly-Ectodermal Dysplasia-Cleft lip/palate syndrome (EEC) syndrome and shown to reduce the transactivation activity of TP63 in a dominant-negative manner. This case represents one of the few instances of immune system involvement in a patient with a TP63 mutation, highlighting the need for further investigation into the immunological aspects of TP63-associated disorders. Our findings suggest that comprehensive immunological evaluation should be considered for patients with TP63 mutations to better understand and manage potential immune dysfunctions.

Keywords: KREC; TP63; TREC; cleft-lip/palate; ectodermal dysplasia; ectrodactyly; lymphopenia; newborn screening.

Publication types

  • Case Reports

MeSH terms

  • Exome Sequencing
  • Humans
  • Infant, Newborn
  • Lymphopenia* / diagnosis
  • Lymphopenia* / genetics
  • Male
  • Mutation
  • Neonatal Screening*
  • Severe Combined Immunodeficiency / diagnosis
  • Severe Combined Immunodeficiency / genetics
  • Transcription Factors* / genetics
  • Tumor Suppressor Proteins* / genetics

Substances

  • TP63 protein, human
  • Tumor Suppressor Proteins
  • Transcription Factors