Quantifiable blood TCR repertoire components associate with immune aging

Nat Commun. 2024 Sep 17;15(1):8171. doi: 10.1038/s41467-024-52522-z.

Abstract

T cell senescence alters the homeostasis of distinct T cell populations and results in decayed adaptive immune protection in older individuals, but a link between aging and dynamic T cell clone changes has not been made. Here, using a newly developed computational framework, Repertoire Functional Units (RFU), we investigate over 6500 publicly available TCR repertoire sequencing samples from multiple human cohorts and identify age-associated RFUs consistently across different cohorts. Quantification of RFU reduction with aging reveals accelerated loss under immunosuppressive conditions. Systematic analysis of age-associated RFUs in clinical samples manifests a potential link between these RFUs and improved clinical outcomes, such as lower ICU admission and reduced risk of complications, during acute viral infections. Finally, patients receiving bone marrow transplantation show a secondary expansion of the age-associated clones upon stem cell transfer from younger donors. Together, our results suggest the existence of a 'TCR clock' that could reflect the immune functions in aging populations.

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Aging* / immunology
  • Bone Marrow Transplantation
  • Cellular Senescence / immunology
  • Female
  • Humans
  • Male
  • Middle Aged
  • Receptors, Antigen, T-Cell* / genetics
  • Receptors, Antigen, T-Cell* / immunology
  • Receptors, Antigen, T-Cell* / metabolism
  • T-Lymphocytes* / immunology
  • Young Adult

Substances

  • Receptors, Antigen, T-Cell