Left atrial compression syndrome secondary to coronary artery perforation: Pathophysiology, diagnosis, and management strategies

Reza Masoomi; Christopher R Burke; Juan Del Cid Fratti; Lorenzo Azzalini; Kathleen E Kearney; William Lombardi

doi:10.1002/ccd.31182

Left atrial compression syndrome secondary to coronary artery perforation: Pathophysiology, diagnosis, and management strategies

Catheter Cardiovasc Interv. 2024 Aug 18. doi: 10.1002/ccd.31182. Online ahead of print.

Authors

Reza Masoomi¹, Christopher R Burke², Juan Del Cid Fratti¹, Lorenzo Azzalini¹, Kathleen E Kearney¹, William Lombardi¹

Affiliations

¹ Division of Cardiology, University of Washington, Seattle, Washington, USA.
² Division of Cardiothoracic Surgery, University of Washington, Seattle, Washington, USA.

PMID: 39154249
DOI: 10.1002/ccd.31182

Abstract

Coronary perforation (CP) poses a significant risk of morbidity and mortality, particularly, in patients with a history of cardiac surgery. The occurrence of loculated pericardial effusion presents distinctive challenges in these postcardiac surgical patients. This study delves into the complexities arising from the formation of loculated pericardial effusions subsequent to CP, with a specific focus on the loculated effusion in the posterior wall leading to left atrial compression syndrome. This analysis is dedicated to elucidating pathophysiology diagnostic and treatment strategies tailored for addressing left atrium compression syndrome, providing invaluable insights into the intricacies of diagnosing, treating, and managing this entity in the postcardiac surgical patient.

Keywords: chronic total occlusion; coronary perforation; left atrial compression syndrome; pericardial effusion.