MGMT protein expression is a reliable predictive biomarker for temozolomide-containing chemotherapy in osteosarcoma

Cancer Sci. 2024 Oct;115(10):3394-3402. doi: 10.1111/cas.16297. Epub 2024 Jul 30.

Abstract

The prognosis of patients with osteosarcoma who experience recurrence or progression (R/P) is extremely poor, and more effective and less toxic therapies are needed. In the current study, the clinical data of osteosarcoma patients who experienced R/P were retrospectively analyzed to verify the reliability of O-6-methylguanine-DNA methyltransferase (MGMT) protein expression or MGMT promoter methylation for predicting the response to off-label temozolomide (TMZ)-containing chemotherapy. Of the 30 evaluable patients, 9 (30%) showed no/low MGMT protein expression, whereas all 16 evaluable patients had unmethylated MGMT promoter irrespective of MGMT protein expression levels. Twenty-three patients received TMZ-containing chemotherapy for measurable lesions (n = 14) or as adjuvant therapy following resection of recurrent lesions (n = 9). Among 14 patients with radiologically measurable lesions, the objective response rate was higher in the MGMT no/low-expression group (50.0%) than in the MGMT intermediate/high-expression group with borderline significance (0%, p = 0.066). The 6-month progression-free survival (PFS) rate in patients with radiologically measurable lesions was significantly higher in the MGMT no/low-expression group (50.0%) than in the MGMT intermediate/high-expression group (0%, p = 0.036). In the multivariate analysis of the 23 patients receiving TMZ-containing chemotherapy, MGMT expression and disease status before TMZ-containing chemotherapy were significantly associated with PFS. No severe adverse effects were observed during TMZ-containing chemotherapy. MGMT protein expression, but not MGMT promoter methylation, could predict a favorable outcome in patients receiving TMZ-containing chemotherapy.

Keywords: O‐6‐methylguanine DNA methyltransferase; biomarker; osteosarcoma; recurrence; temozolomide.

MeSH terms

  • Adolescent
  • Adult
  • Antineoplastic Agents, Alkylating / therapeutic use
  • Biomarkers, Tumor* / genetics
  • Biomarkers, Tumor* / metabolism
  • Bone Neoplasms* / drug therapy
  • Bone Neoplasms* / genetics
  • Bone Neoplasms* / mortality
  • Bone Neoplasms* / pathology
  • Child
  • DNA Methylation*
  • DNA Modification Methylases* / genetics
  • DNA Modification Methylases* / metabolism
  • DNA Repair Enzymes* / genetics
  • DNA Repair Enzymes* / metabolism
  • Female
  • Humans
  • Male
  • Middle Aged
  • Neoplasm Recurrence, Local / drug therapy
  • Neoplasm Recurrence, Local / genetics
  • Neoplasm Recurrence, Local / pathology
  • Osteosarcoma* / drug therapy
  • Osteosarcoma* / genetics
  • Osteosarcoma* / mortality
  • Osteosarcoma* / pathology
  • Prognosis
  • Promoter Regions, Genetic*
  • Retrospective Studies
  • Temozolomide* / therapeutic use
  • Tumor Suppressor Proteins* / genetics
  • Tumor Suppressor Proteins* / metabolism
  • Young Adult

Substances

  • Temozolomide
  • MGMT protein, human
  • DNA Repair Enzymes
  • Tumor Suppressor Proteins
  • DNA Modification Methylases
  • Biomarkers, Tumor
  • Antineoplastic Agents, Alkylating