The Role of Diet and the Gut Microbiota in Reactive Aggression and Adult ADHD-An Exploratory Analysis

Nutrients. 2024 Jul 9;16(14):2174. doi: 10.3390/nu16142174.

Abstract

Attention-deficit/hyperactivity disorder (ADHD) is a common neurodevelopmental condition, of-ten persistent into adulthood and accompanied by reactive aggression. Associations of diet and the gut-microbiome with ADHD as well as emotional behaviors suggest potential clinical rele-vance of both. However, studies on diet and the gut-microbiome in human reactive aggression are lacking, and should investigate the interaction between diet and the gut-microbiome leading to behavioral changes to assess their potential clinical relevance. In this study, we investigated the interaction of diet and gut-microbiota with adult ADHD and reactive aggression in 77 adults with ADHD and 76 neurotypical individuals. We studied the relationships of ADHD and reactive ag-gression with dietary patterns, bacterial community and taxonomic differences of 16S-sequenced fecal microbiome samples, and potential mediating effects of bacterial genus abundance on signifi-cant diet-behavior associations. The key findings include: (1) An association of high-energy intake with reactive aggeression scores (pFDR = 4.01 × 10-02); (2) Significant associations of several genera with either reactive aggression or ADHD diagnosis with no overlap; and (3) No significant mediation effects of the selected genera on the association of reactive aggression with the high-energy diet. Our results suggest that diet and the microbiome are linked to reactive aggression and/or ADHD individually, and highlight the need to further study the way diet and the gut-microbiome inter-act.

Keywords: ADHD; diet; gut microbiota; reactive aggression.

MeSH terms

  • Adult
  • Aggression*
  • Attention Deficit Disorder with Hyperactivity* / microbiology
  • Diet*
  • Energy Intake
  • Feces / microbiology
  • Female
  • Gastrointestinal Microbiome*
  • Humans
  • Male
  • Middle Aged
  • Young Adult

Grants and funding

We acknowledge funding from the Netherlands Organization for Scientific Research (NWO), i.e., the Veni Innovation Program (grant 016-196-115 to M.H.) and the Vici Innovation Program (grant 016–130-669 to B.F.). The work was also supported by the European College of Neuropsychopharmacology (ECNP) Network “ADHD Across the Lifespan”. B.J. and B.F. were also supported by funding from the European Community’s Horizon 2020 Programme (H2020/2014–2020) under grant agreement n° 847879 (PRIME). Concurrently, the research contributing to these results was also supported by funding from the European Community’s Horizon 2020 research and innovation program, specifically through the Eat2beNICE project (grant agreement no. 728018), the CANDY project (grant agreement no. 847818) and the DISCOvERIE project (grant agreement no. 848228).