Abstract
Giant congenital melanocytic nevi (GCMN) can be cosmetically significant and can lead to melanoma. There is no standard pharmacologic treatment for GCMN. We present the case of an 8-year-old female with kaposiform lymphangiomatosis caused by an NRAS mutation whose nevus spilus-type GCMN improved on oral selumetinib.
Keywords:
MEK inhibitor; NRAS; kaposiform lymphangiomatosis; nevus spilus.
© 2024 The Author(s). Pediatric Dermatology published by Wiley Periodicals LLC.
MeSH terms
-
Administration, Oral
-
Benzimidazoles* / therapeutic use
-
Child
-
Female
-
GTP Phosphohydrolases / genetics
-
Humans
-
Membrane Proteins / genetics
-
Mutation
-
Nevus, Pigmented* / congenital
-
Nevus, Pigmented* / drug therapy
-
Nevus, Pigmented* / genetics
-
Nevus, Pigmented* / pathology
-
Skin Neoplasms* / congenital
-
Skin Neoplasms* / drug therapy
-
Skin Neoplasms* / genetics
-
Skin Neoplasms* / pathology
Substances
-
Benzimidazoles
-
AZD 6244
-
NRAS protein, human
-
GTP Phosphohydrolases
-
Membrane Proteins
Supplementary concepts
-
Melanocytic nevus syndrome, congenital