Anti-inflammatory, Antioxidative, and Moisturizing Effects of Oxyceros horridus Lour. Ethanol Extract in Human Keratinocytes via the p38 Signaling Pathway

Chem Biodivers. 2024 Apr;21(4):e202301791. doi: 10.1002/cbdv.202301791. Epub 2024 Mar 13.

Abstract

Skin is the largest and outermost organ in the human body; it serves as a vital defense mechanism against various external threats. Therefore, it is crucial to maintain its health through protection against harmful substances and adequate moisture levels. This study investigates the anti-inflammatory, antioxidant, and moisturizing properties of Oxyceros horridus Lour. (Oh-EE) in human keratinocytes. Oh-EE demonstrates potent antioxidant activity and effectively protects against oxidative stress induced by external stimuli such as UVB radiation and H2O2. Additionally, it exhibits significant anti-inflammatory effects proven by its ability to downregulate the expression of pro-inflammatory cytokines, namely COX-2 and IL-6. The study also explores the involvement of the AP-1 pathway, highlighting the ability of Oh-EE to suppress the expression of p38 and its upstream regulator, MKK3/6, under UVB-induced conditions. Interestingly, Oh-EE can activate the AP-1 pathway in the absence of external triggers. Furthermore, Oh-EE enhances skin moisture by upregulating the expression of key genes involved in skin hydration, namely HAS3 and FLG. These findings underscore the potential of Oh-EE as a versatile ingredient in skincare formulations, providing a range of skin benefits. Further research is warranted to comprehensively understand the underlying mechanisms through which Oh-EE exerts its effects.

Keywords: Oxyceros horridus; UVB; anti-inflammation; antioxidant; moisturizing; skin.

MeSH terms

  • Anti-Inflammatory Agents / metabolism
  • Anti-Inflammatory Agents / pharmacology
  • Antioxidants* / metabolism
  • Antioxidants* / pharmacology
  • Ethanol* / pharmacology
  • Humans
  • Hydrogen Peroxide / pharmacology
  • Keratinocytes
  • Signal Transduction
  • Transcription Factor AP-1 / metabolism
  • Transcription Factor AP-1 / pharmacology

Substances

  • Antioxidants
  • Ethanol
  • Hydrogen Peroxide
  • Transcription Factor AP-1
  • Anti-Inflammatory Agents