Dietary-derived vitamin B12 protects Caenorhabditis elegans from thiol-reducing agents

BMC Biol. 2022 Oct 8;20(1):228. doi: 10.1186/s12915-022-01415-y.

Abstract

Background: One-carbon metabolism, which includes the folate and methionine cycles, involves the transfer of methyl groups which are then utilised as a part of multiple physiological processes including redox defence. During the methionine cycle, the vitamin B12-dependent enzyme methionine synthetase converts homocysteine to methionine. The enzyme S-adenosylmethionine (SAM) synthetase then uses methionine in the production of the reactive methyl carrier SAM. SAM-binding methyltransferases then utilise SAM as a cofactor to methylate proteins, small molecules, lipids, and nucleic acids.

Results: We describe a novel SAM methyltransferase, RIPS-1, which was the single gene identified from forward genetic screens in Caenorhabditis elegans looking for resistance to lethal concentrations of the thiol-reducing agent dithiothreitol (DTT). As well as RIPS-1 mutation, we show that in wild-type worms, DTT toxicity can be overcome by modulating vitamin B12 levels, either by using growth media and/or bacterial food that provide higher levels of vitamin B12 or by vitamin B12 supplementation. We show that active methionine synthetase is required for vitamin B12-mediated DTT resistance in wild types but is not required for resistance resulting from RIPS-1 mutation and that susceptibility to DTT is partially suppressed by methionine supplementation. A targeted RNAi modifier screen identified the mitochondrial enzyme methylmalonyl-CoA epimerase as a strong genetic enhancer of DTT resistance in a RIPS-1 mutant. We show that RIPS-1 is expressed in the intestinal and hypodermal tissues of the nematode and that treating with DTT, β-mercaptoethanol, or hydrogen sulfide induces RIPS-1 expression. We demonstrate that RIPS-1 expression is controlled by the hypoxia-inducible factor pathway and that homologues of RIPS-1 are found in a small subset of eukaryotes and bacteria, many of which can adapt to fluctuations in environmental oxygen levels.

Conclusions: This work highlights the central importance of dietary vitamin B12 in normal metabolic processes in C. elegans, defines a new role for this vitamin in countering reductive stress, and identifies RIPS-1 as a novel methyltransferase in the methionine cycle.

Keywords: DTT; Methionine; Methyltransferase; Reductive stress; Vitamin B12.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase / genetics
  • 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase / metabolism
  • Animals
  • Caenorhabditis elegans / metabolism
  • Carbon / metabolism
  • Dithiothreitol / metabolism
  • Folic Acid / metabolism
  • Homocysteine / metabolism
  • Hydrogen Sulfide* / metabolism
  • Ligases / metabolism
  • Lipids
  • Mercaptoethanol / metabolism
  • Methionine / metabolism
  • Methyltransferases / genetics
  • Methyltransferases / metabolism
  • Nucleic Acids*
  • Oxygen / metabolism
  • Reducing Agents / metabolism
  • S-Adenosylmethionine / metabolism
  • Sulfhydryl Compounds / metabolism
  • Vitamin B 12 / metabolism
  • Vitamin B 12 / pharmacology
  • Vitamins / metabolism

Substances

  • Lipids
  • Nucleic Acids
  • Reducing Agents
  • Sulfhydryl Compounds
  • Vitamins
  • Homocysteine
  • Mercaptoethanol
  • Carbon
  • S-Adenosylmethionine
  • Folic Acid
  • Methionine
  • Methyltransferases
  • 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase
  • Ligases
  • Vitamin B 12
  • Oxygen
  • Dithiothreitol
  • Hydrogen Sulfide