Reduced Graphene Oxides Modulate the Expression of Cell Receptors and Voltage-Dependent Ion Channel Genes of Glioblastoma Multiforme

Int J Mol Sci. 2021 Jan 6;22(2):515. doi: 10.3390/ijms22020515.

Abstract

The development of nanotechnology based on graphene and its derivatives has aroused great scientific interest because of their unusual properties. Graphene (GN) and its derivatives, such as reduced graphene oxide (rGO), exhibit antitumor effects on glioblastoma multiforme (GBM) cells in vitro. The antitumor activity of rGO with different contents of oxygen-containing functional groups and GN was compared. Using FTIR (fourier transform infrared) analysis, the content of individual functional groups (GN/exfoliation (ExF), rGO/thermal (Term), rGO/ammonium thiosulphate (ATS), and rGO/ thiourea dioxide (TUD)) was determined. Cell membrane damage, as well as changes in the cell membrane potential, was analyzed. Additionally, the gene expression of voltage-dependent ion channels (clcn3, clcn6, cacna1b, cacna1d, nalcn, kcne4, kcnj10, and kcnb1) and extracellular receptors was determined. A reduction in the potential of the U87 glioma cell membrane was observed after treatment with rGO/ATS and rGO/TUD flakes. Moreover, it was also demonstrated that major changes in the expression of voltage-dependent ion channel genes were observed in clcn3, nalcn, and kcne4 after treatment with rGO/ATS and rGO/TUD flakes. Furthermore, the GN/ExF, rGO/ATS, and rGO/TUD flakes significantly reduced the expression of extracellular receptors (uPar, CD105) in U87 glioblastoma cells. In conclusion, the cytotoxic mechanism of rGO flakes may depend on the presence and types of oxygen-containing functional groups, which are more abundant in rGO compared to GN.

Keywords: cell membrane receptor; glioblastoma multiforme; graphene; membrane potential; reduced graphene oxide; voltage-gated ion channel.

MeSH terms

  • Cell Line, Tumor
  • Cells
  • Chloride Channels / genetics*
  • Chloride Channels / metabolism
  • Gene Expression Regulation, Neoplastic / drug effects*
  • Glioblastoma / genetics*
  • Glioblastoma / metabolism
  • Glioblastoma / pathology
  • Graphite / chemistry
  • Graphite / pharmacology*
  • Humans
  • Ion Channels / genetics*
  • Ion Channels / metabolism
  • Membrane Potentials / drug effects
  • Membrane Proteins / genetics*
  • Membrane Proteins / metabolism
  • Microscopy, Electron, Scanning
  • Oxidation-Reduction
  • Potassium Channels, Voltage-Gated / genetics*
  • Potassium Channels, Voltage-Gated / metabolism
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • Receptors, Cell Surface / genetics*
  • Receptors, Cell Surface / metabolism
  • Spectroscopy, Fourier Transform Infrared

Substances

  • Chloride Channels
  • ClC-3 channel
  • Ion Channels
  • KCNE4 protein, human
  • Membrane Proteins
  • NALCN protein, human
  • Potassium Channels, Voltage-Gated
  • RNA, Messenger
  • Receptors, Cell Surface
  • graphene oxide
  • Graphite