Mechanisms of drug resistance mediated by long non-coding RNAs in non-small-cell lung cancer

Cancer Gene Ther. 2021 Apr;28(3-4):175-187. doi: 10.1038/s41417-020-00214-3. Epub 2020 Aug 25.

Abstract

Non-small-cell lung cancer (NSCLC) is the most prevalent form of lung cancer and has a poor five-year survival rate of 15%. Chemotherapy and targeted therapies have significantly improved patients' prognosis. Nevertheless, after a successful initial response, some patients relapse when cancer cells become resistant to drug treatments, representing an important clinical limitation. Therefore, investigating the mechanisms of drug resistance is of significant importance. Recently, considerable attention has been given to long non-coding RNAs (lncRNAs), a heterogeneous class of regulatory molecules that play essential roles in tumorigenesis by modulating genes and signalling pathways involved in cell growth, metastasis and drug response. In this article, we review recent research findings on the role of lncRNAs in drug resistance in NSCLC, highlighting their mechanisms of action.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Carcinoma, Non-Small-Cell Lung / genetics
  • Drug Resistance, Neoplasm / genetics*
  • Lung Neoplasms / genetics*
  • RNA, Long Noncoding / metabolism*

Substances

  • RNA, Long Noncoding