Autism in neurofibromatosis type 1: misuse of covariance to dismiss autistic trait burden
Dev Med Child Neurol
.
2021 Feb;63(2):233-234.
doi: 10.1111/dmcn.14653.
Epub 2020 Aug 20.
Authors
Stephanie M Morris
1
,
Maria T Acosta
2
,
Shruti Garg
3
,
Jonathan Green
3
,
Eric Legius
4
,
Kathryn North
5
,
Jonathan M Payne
5
,
Lauren A Weiss
6
,
John N Constantino
7
,
David H Gutmann
1
Affiliations
1
Department of Neurology, Washington University School of Medicine, St. Louis, MO, USA.
2
National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, USA.
3
Royal Manchester Children's Hospital, Manchester, UK.
4
Department of Human Genetics, KU Leuven, Leuven, Belgium.
5
Department of Pediatrics, Murdoch Children's Research Institute, University of Melbourne, Melbourne, Vic, Australia.
6
Department of Psychiatry, Institute for Human Genetics, University of California, San Francisco, CA, USA.
7
Department of Psychiatry, Washington University School of Medicine, St. Louis, MO, USA.
PMID:
32815557
DOI:
10.1111/dmcn.14653
No abstract available
Publication types
Letter
Comment
MeSH terms
Attention Deficit Disorder with Hyperactivity*
Autism Spectrum Disorder* / complications
Autistic Disorder* / etiology
Child
Humans
Neurofibromatosis 1* / complications
Phenotype