Evi-1 Influence on Clinical Progress of Colorectal Cancer Patients

Ann Clin Lab Sci. 2020 May;50(3):354-360.

Abstract

Objective: Aim of this study is to assess effects of Evi-1 on regulation of c-Jun N-terminal kinase (JNK)/c-Jun pathway on colorectal cancer and the relationship of their expression with clinicopathological characteristics and prognosis.

Methods: The clinical data of 394 CRC patients and the mRNA expression of Evi-1 were downloaded from The Cancer Genome Atlas (TCGA). Immunohistochemical (IHC) method was used to detect the protein expression of Evi-1, JNK and c-Jun in CRC tissues and adjacent normal tissues.

Results: The TCGA dataset demonstrated that Evi-1 mRNA was upregulated in CRC. Evi-1 mRNA expression was significantly correlated with lymphatic metastasis, T stage, distant metastasis and TNM stage. IHC staining showed that Remmele immunoreactive Score (IRS) of Evi-1, JNK and c-Jun were highly expressed in CRC tissues compared with normal tissues adjacent to cancer. Evi-1 in CRC tissues was negatively correlated with JNK and c-Jun. Overexpression of Evi-1, JNK and c-Jun was significantly correlated with the degree of differentiation, T staging, lymphatic metastasis, distant metastasis and TNM classification.

Conclusion: The present study shows that Evi-1 expression is negatively correlated with JNK and c-Jun expression, and closely related to some clinical data. Further, Evi-1 combined with JNK and c-Jun can be used for adverse prognosis of CRC.

Keywords: Apoptosis; Colorectal cancer; Evi-1; Immunochemistry; JNK.

MeSH terms

  • Adult
  • Aged
  • Biomarkers, Tumor / metabolism
  • Cell Differentiation / genetics
  • Colonic Neoplasms / genetics
  • Colonic Neoplasms / pathology
  • Colorectal Neoplasms / genetics*
  • Colorectal Neoplasms / metabolism
  • Colorectal Neoplasms / pathology*
  • Databases, Genetic
  • Female
  • Gene Expression / genetics
  • Gene Expression Regulation, Neoplastic / genetics
  • Genes, jun / genetics
  • Humans
  • JNK Mitogen-Activated Protein Kinases / genetics
  • JNK Mitogen-Activated Protein Kinases / metabolism
  • Lymphatic Metastasis / genetics
  • MDS1 and EVI1 Complex Locus Protein / genetics
  • MDS1 and EVI1 Complex Locus Protein / metabolism*
  • Male
  • Middle Aged
  • Neoplasm Staging / methods
  • Prognosis
  • Retrospective Studies

Substances

  • Biomarkers, Tumor
  • MDS1 and EVI1 Complex Locus Protein
  • MECOM protein, human
  • JNK Mitogen-Activated Protein Kinases