Association of genomic variants at the human leukocyte antigen locus with cervical cancer risk, HPV status and gene expression levels

Int J Cancer. 2020 Nov 1;147(9):2458-2468. doi: 10.1002/ijc.33171. Epub 2020 Jul 10.

Abstract

The human leukocyte antigen (HLA) locus on chromosome 6 has been reported to be associated with cervical cancer. We investigated two independent single-nucleotide polymorphisms in a large case-control series of cervical dysplasia and carcinoma that has been newly established by the German Cervigen Consortium, comprising a total of 2481 cases and 1556 healthy females. We find significant associations for both variants, rs9272117 at HLA-DQA1 and rs2844511 at MICA and HCP5, with cervical disease. Both variants showed evidence of association with invasive cervical cancer (rs9272117: OR 0.89, 95% CI 0.79-0.99, P = .036; rs2844511: OR 1.17, 95% CI 1.04-1.31, P = .008) and with high-grade dysplasia (rs9272117: OR 0.78, 95% CI 0.70-0.87, P = 7.1 × 10-6 ; rs2844511: OR 1.13, 95% CI 1.01-1.26, P = .035), as well as in a combined analysis of both groups (rs9272117: OR 0.83, 95% CI 0.75-0.91, P = 6.9 × 10-5 ; rs2844511: OR 1.14, 95% CI 1.04-1.26, P = .005). Variant rs2844511, but not rs9272117, also showed modest evidence of association with low-grade dysplasia (OR 1.26, 95% CI 1.04-1.54, P = .019). In case-only analyses, rs2844511 tended to predict HPV status (P = .044) and rs9272117 tended to associate with HPV16 (P = .022). RNA studies in cervical samples showed a significant correlation in the transcript levels of MICA, HCP5 and HLA-DQA1, suggesting extensive co-regulation. All three genes were upregulated in HPV16-positive samples. In stratified analyses, rs9272117 was associated with HLA-DQA1 levels, specifically in HPV-positive samples, while rs2844511 was associated with MICA and HCP5 levels. The risk allele of rs2844511 was required for correlations between MICA or HCP5 with HLA-DQA1. Altogether, our results support 6p21.32-33 as the first consistent cervical cancer susceptibility locus and provide evidence for a link between genetic risk variants, HPV16 status and transcript levels of HLA-DQA1, HCP5 and MICA, which may contribute to tumor immune evasion.

Keywords: HPV infection; association study; cervical malignancy; eQTL; single nucleotide polymorphism.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Aged, 80 and over
  • Alleles
  • Case-Control Studies
  • Cervix Uteri / immunology
  • Cervix Uteri / pathology*
  • Cervix Uteri / virology
  • Female
  • Gene Expression Regulation, Neoplastic / immunology
  • Genetic Loci
  • Genetic Predisposition to Disease
  • Germany / epidemiology
  • HLA Antigens / genetics*
  • HLA Antigens / immunology
  • Human papillomavirus 16 / immunology
  • Human papillomavirus 16 / isolation & purification
  • Humans
  • Middle Aged
  • Papillomavirus Infections / epidemiology*
  • Papillomavirus Infections / genetics
  • Papillomavirus Infections / immunology
  • Papillomavirus Infections / virology
  • Polymorphism, Single Nucleotide
  • Tumor Escape / genetics
  • Up-Regulation / immunology
  • Uterine Cervical Dysplasia / epidemiology*
  • Uterine Cervical Dysplasia / genetics
  • Uterine Cervical Dysplasia / immunology
  • Uterine Cervical Dysplasia / virology
  • Uterine Cervical Neoplasms / epidemiology*
  • Uterine Cervical Neoplasms / genetics
  • Uterine Cervical Neoplasms / immunology
  • Uterine Cervical Neoplasms / virology
  • Young Adult

Substances

  • HLA Antigens