UVC-Emitting LuPO4:Pr3+ Nanoparticles Decrease Radiation Resistance of Hypoxic Cancer Cells

Radiat Res. 2020 Jan;193(1):82-87. doi: 10.1667/RR15491.1. Epub 2019 Nov 18.

Abstract

Radiation-resistant hypoxic tumor areas continue to present a major limitation for successful tumor treatment. To overcome this radiation resistance, an oxygen-independent treatment is proposed using UVC-emitting LuPO4:Pr3+ nanoparticles (NPs) and X rays. The uptake of the NPs as well as their effect on cell proliferation was investigated on A549 lung cancer cells by using inverted time-lapse microscopy and transmission electron microscopy. Furthermore, cytotoxicity of the combined treatment of X rays and LuPO4:Pr3+ NPs was assessed under normoxic and hypoxic conditions using the colony formation assay. Transmission electron microscopy (TEM) images showed no NP uptake after 3 h, whereas after 24 h incubation an uptake of NPs was documented. LuPO4:Pr3+ NPs alone caused a concentration-independent cell growth delay within the first 60 h of incubation. The combined treatment with UVC-emitting NPs and X rays reduced the radiation resistance of hypoxic cells by a factor of two to the level of cells under normoxic condition. LuPO4:Pr3+ NPs cause an early growth delay but no cytotoxicity for the tested concentration. The combination of these NPs with X rays increases cytotoxicity of normoxic and hypoxic cancer cells. Hypoxic cells become sensitized to normoxic cell levels.

MeSH terms

  • A549 Cells
  • Cell Proliferation / drug effects
  • Cell Proliferation / radiation effects
  • Humans
  • Lutetium*
  • Nanoparticles*
  • Praseodymium*
  • Radiation Tolerance / drug effects*
  • Radiation Tolerance / radiation effects
  • Tumor Hypoxia / radiation effects*
  • Ultraviolet Rays*

Substances

  • LuPO4:Pr(3+)
  • Lutetium
  • Praseodymium