Abstract
Checkpoint blockade (CPB) therapy can elicit durable clinical responses by reactivating an exhausted immune response. However, response rates remain limited, likely secondary to a lack of a tumor-reactive immune infiltrate. Chimeric antigen receptor (CAR) T cells may provide the necessary tumor-targeting immune infiltrate and a highly specific antitumor immune response. This can be further amplified by the addition of CPB agents, which serve to counteract the immune inhibitory environment undermining optimal CAR T cell efficacy. Herein, we review preclinical and clinical combination therapy with CAR T cells and CPB agents, with a focus on solid tumor malignancies.
Keywords:
CAR T cell; PD-1; checkpoint blockade; chimeric antigen receptor; immunotherapy.
Copyright © 2019 Elsevier Inc. All rights reserved.
Publication types
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Research Support, N.I.H., Extramural
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Research Support, U.S. Gov't, Non-P.H.S.
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Review
MeSH terms
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Animals
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Antineoplastic Agents, Immunological / pharmacology
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Antineoplastic Agents, Immunological / therapeutic use*
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Combined Modality Therapy / methods
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Costimulatory and Inhibitory T-Cell Receptors / antagonists & inhibitors*
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Costimulatory and Inhibitory T-Cell Receptors / immunology
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Costimulatory and Inhibitory T-Cell Receptors / metabolism
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Disease Models, Animal
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Humans
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Immunotherapy, Adoptive / methods*
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Neoplasms / immunology
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Neoplasms / pathology
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Neoplasms / therapy*
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Randomized Controlled Trials as Topic
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Receptors, Chimeric Antigen / immunology*
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Treatment Outcome
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Tumor Escape / drug effects
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Tumor Escape / immunology
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Tumor Microenvironment / drug effects
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Tumor Microenvironment / immunology
Substances
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Antineoplastic Agents, Immunological
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Costimulatory and Inhibitory T-Cell Receptors
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Receptors, Chimeric Antigen