The Phosphatase PRL-3 Is Involved in Key Steps of Cancer Metastasis

J Mol Biol. 2019 Aug 9;431(17):3056-3067. doi: 10.1016/j.jmb.2019.06.008. Epub 2019 Jun 14.

Abstract

PRL-3 belongs to the PRL phosphatase family. Its physiological role remains unclear, but many studies have identified that PRL-3 is a marker of cancer progression and shown it to be associated with metastasis. Evidence implicating PRL-3 in various elements of the metastatic process, such as the cell cycle, survival, angiogenesis, adhesion, cytoskeleton remodeling, EMT, motility and invasion, has been reported. Furthermore, several molecules acting as direct or indirect substrates have been identified. However, this information was obtained in many different studies, and it remains difficult to see the larger picture. We therefore systematically collected the published information together and used it to develop a comprehensive signaling network map. By analyzing this network map, we were able to retrieve the signaling pathways via which PRL-3 governs the key steps of the metastatic process in cancer. In this review, we summarize current knowledge of the role of PRL-3 in cancer and the molecular mechanisms involved. We also provide the web-based open-source PRL-3 signaling network map, for use in further studies.

Keywords: metastasis; phosphatase; signaling pathways; systems biology; tumorigenesis.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Apoptosis
  • Carcinogenesis / metabolism*
  • Cell Adhesion
  • Cell Cycle
  • Cell Line, Tumor
  • Cell Movement
  • Cytoskeleton
  • Disease Progression
  • Humans
  • Neoplasm Metastasis*
  • Neoplasm Proteins / metabolism*
  • Protein Tyrosine Phosphatases / metabolism*
  • Signal Transduction
  • Systems Biology

Substances

  • Neoplasm Proteins
  • PTP4A3 protein, human
  • Protein Tyrosine Phosphatases