Diclofenac-Derived Hybrids for Treatment of Actinic Keratosis and Squamous Cell Carcinoma

Molecules. 2019 May 9;24(9):1793. doi: 10.3390/molecules24091793.

Abstract

In this work, hybrid compounds 1-4 obtained by conjugation of the non-steroidal anti-inflammatory drug diclofenac, with natural molecules endowed with antioxidant and antiproliferative activity were prepared. The antiproliferative activity of these hybrids was evaluated on immortalized human keratinocyte (HaCaT) cells stimulated with epidermal growth factor (EGF), an actinic keratosis (AK) model, and on human squamous cell carcinoma (SCC) cells (A431). Hybrid 1 presented the best activity in both cell models. Self-assembling surfactant nanomicelles have been chosen as the carrier to drive the hybrid 1 into the skin; the in vitro permeation through and penetration into pig ear skin have been evaluated. Among the nanostructured formulations tested, Nano3Hybrid20 showed a higher tendency of the hybrid 1 to be retained in the skin rather than permeating it, with a desirable topical and non-systemic action. On these bases, hybrid 1 may represent an attractive lead scaffold for the development of new treatments for AK and SCC.

Keywords: actinic keratosis; antiproliferative activity; diclofenac; hybrid; in vitro skin permeation/penetration; nanomicelles; squamous cell carcinoma.

MeSH terms

  • Animals
  • Carcinoma, Squamous Cell / drug therapy*
  • Carcinoma, Squamous Cell / pathology
  • Cell Line
  • Cell Proliferation / drug effects
  • Cell Survival / drug effects
  • Diclofenac / chemical synthesis
  • Diclofenac / chemistry
  • Diclofenac / pharmacology
  • Diclofenac / therapeutic use*
  • Humans
  • Inhibitory Concentration 50
  • Keratosis, Actinic / drug therapy*
  • Keratosis, Actinic / pathology
  • Micelles
  • Nanoparticles / chemistry
  • Particle Size
  • Skin Neoplasms / drug therapy*
  • Skin Neoplasms / pathology
  • Swine

Substances

  • Micelles
  • Diclofenac