Follicle-stimulating hormone peptide-conjugated nanoparticles for targeted shRNA delivery lead to effective gro-α silencing and antitumor activity against ovarian cancer

Drug Deliv. 2018 Nov;25(1):576-584. doi: 10.1080/10717544.2018.1440667. Epub 2018 Feb 20.

Abstract

The distinct hormone molecules and receptors, such as follicle-stimulating hormone receptor (FSHR) in ovarian cancer, provide opportunities for more precisely targeted therapy. We previously developed FSHR-mediated nanoparticles and found that FSH peptides on the surface of nanoparticles improved the delivery of short interfering RNA (siRNA) into ovarian cancer cells. However, the high toxicity of the nanoparticles and the transient silencing of the siRNA in vivo limited further study. Here, we developed FSH peptide-conjugated nanoparticles with an increased amount of polyethylene glycol (PEG) grafting and encapsulated short hairpin RNA (shRNA) to silence the target gene, growth-regulated oncogene α (gro-α). The nanoparticle complexes exhibited good stability over three weeks. Expression of the target gene, gro-α, was significantly down-regulated by gro-α shRNA-loaded nanoparticles conjugated with FSH peptides (FSH33-G-NP) in FSHR-positive HEY cells. Cell proliferation, migration, and invasion were also inhibited by FSH33-G-NP. Tumor growth was delayed significantly in the mice treated with FSH33-G-NP. No significant loss of body weight or severe toxic effects were observed in any groups. In conclusion, gro-α shRNA-loaded nanoparticles conjugated with FSH peptides overcame the drawbacks of the in vivo application of RNAi therapeutics and polymer-based nanocarriers and showed safe antitumor efficacy. Our study might contribute to the application of FSHR-based targeted therapy and imaging in cancer.

Keywords: Ovarian carcinoma; follicle-stimulating hormone; growth-regulated oncogene α; short hairpin RNA; targeted therapy.

MeSH terms

  • Animals
  • Antineoplastic Agents / administration & dosage*
  • Cell Line, Tumor
  • Chemokine CXCL1 / administration & dosage*
  • Chemokine CXCL1 / biosynthesis
  • Chemokine CXCL1 / genetics
  • Female
  • Follicle Stimulating Hormone / administration & dosage*
  • Gene Silencing / drug effects
  • Gene Silencing / physiology
  • Gene Transfer Techniques
  • Humans
  • Mice
  • Mice, Inbred BALB C
  • Mice, Nude
  • Nanoparticles / administration & dosage*
  • Ovarian Neoplasms / drug therapy*
  • Ovarian Neoplasms / genetics
  • Ovarian Neoplasms / pathology
  • RNA, Small Interfering / administration & dosage*
  • RNA, Small Interfering / genetics
  • Xenograft Model Antitumor Assays

Substances

  • Antineoplastic Agents
  • CXCL1 protein, human
  • Chemokine CXCL1
  • RNA, Small Interfering
  • Follicle Stimulating Hormone

Grants and funding

This work was supported by the National Natural Sciences Foundation of China under [Grant No. 81472424]; the National Key R&D Program of China under [Grant No. 2016YFC1303100]; the Youth Talent Foundation of Shanghai Health System under [Grant No. XYQ2011054].