SOX7 Suppresses Wnt Signaling by Disrupting β-Catenin/BCL9 Interaction

DNA Cell Biol. 2018 Feb;37(2):126-132. doi: 10.1089/dna.2017.3866. Epub 2017 Dec 22.

Abstract

The Wnt signaling is involved in angiogenesis and tumor development. β-catenin is the core component of the Wnt pathway, which mediates oncogenic transcription and regulated by a series of proteins. Sex-determining region Y-box 7 (SOX7) is a member of high-mobility-group transcription factor family, which inhibits oncogenic Wnt signaling in lots of tumor cells with unknown mechanism. By coimmunoprecipitation (co-IP) and super Topflash reporter assay, SOX7 can bind β-catenin and inhibit β-catenin/T cell factor (TCF)-mediated transcription. Meanwhile, B cell lymphoma 9 (BCL9) drives Wnt signaling path through direct binding-mediated β-catenin. Finally, we found that SOX7 inhibits oncogenic β-catenin-mediated transcription by disrupting the β-catenin/BCL9 interaction. Mechanistically, SOX7 compete with BCL9 to bind β-catenin. Our results show SOX7 inhibited Wnt signaling as suppressor and could be an important target for anticancer therapy.

Keywords: BCL9; SOX7; Wnt signaling; cancer; β-catenin.

MeSH terms

  • Cell Proliferation
  • HEK293 Cells
  • Humans
  • Neoplasm Proteins / metabolism*
  • Protein Binding
  • SOXF Transcription Factors / physiology*
  • Transcription Factors
  • Transcription, Genetic
  • Wnt Signaling Pathway
  • beta Catenin / metabolism*

Substances

  • BCL9 protein, human
  • CTNNB1 protein, human
  • Neoplasm Proteins
  • SOX7 protein, human
  • SOXF Transcription Factors
  • Transcription Factors
  • beta Catenin