Artificial human antigen-presenting cells are superior to dendritic cells at inducing cytotoxic T-cell responses

Immunology. 2017 Nov;152(3):462-471. doi: 10.1111/imm.12783. Epub 2017 Jul 27.

Abstract

Peptide recognition through the MHC class I molecule by cytotoxic T lymphocytes (CTLs) leads to the killing of cancer cells. A potential challenge for T-cell immunotherapy is that dendritic cells (DCs) are exposed to the MHC class I-peptide complex for an insufficient amount of time. To improve tumour antigen presentation to T cells and thereby initiate a more effective T-cell response, we generated artificial antigen-presenting cells (aAPCs) by incubating human immature DCs (imDCs) with poly(lactic-co-glycolic) acid nanoparticles (PLGA-NPs) encapsulating tumour antigenic peptides, followed by maturation with lipopolysaccharide. Tumour antigen-specific CTLs were then induced using either peptide-loaded mature DCs (mDCs) or aAPCs, and their activities were analysed using both ELISpot and cytotoxicity assays. We found that the aAPCs induced significantly stronger tumour antigen-specific CTL responses than the controls, which included both mDCs and aAPCs loaded with empty nanoparticles. Moreover, frozen CTLs that were generated by exposure to aAPCs retained the capability to eradicate HLA-A2-positive tumour antigen-bearing cancer cells. These results indicated that aAPCs are superior to DCs when inducing the CTL response because the former are capable of continuously presenting tumour antigens to T cells in a sustained manner. The development of aAPCs with PLGA-NPs encapsulating tumour antigenic peptides is a promising approach for the generation of effective CTL responses in vitro and warrants further assessments in clinical trials.

Keywords: artificial antigen-presenting cells; cytotoxic T lymphocytes; dendritic cells; peptide; poly(lactide-co-glycolide) acid.

MeSH terms

  • Antigen Presentation*
  • Cancer Vaccines / chemistry
  • Cancer Vaccines / immunology
  • Cancer Vaccines / pharmacology*
  • Cell Survival / drug effects
  • Cytotoxicity, Immunologic / drug effects*
  • Delayed-Action Preparations
  • Dendritic Cells / drug effects*
  • Dendritic Cells / immunology
  • Dendritic Cells / metabolism
  • Drug Compounding
  • Drug Liberation
  • Humans
  • Inhibitor of Apoptosis Proteins / chemistry
  • Inhibitor of Apoptosis Proteins / immunology
  • Inhibitor of Apoptosis Proteins / pharmacology*
  • Kinetics
  • Lactic Acid / chemistry*
  • Lipopolysaccharides / immunology
  • Lipopolysaccharides / pharmacology*
  • MART-1 Antigen / chemistry
  • MART-1 Antigen / immunology
  • MART-1 Antigen / pharmacology*
  • MCF-7 Cells
  • Nanoparticles*
  • Neoplasms / immunology
  • Neoplasms / metabolism
  • Neoplasms / pathology
  • Neoplasms / therapy*
  • Peptide Fragments / chemistry
  • Peptide Fragments / immunology
  • Peptide Fragments / pharmacology*
  • Polyglycolic Acid / chemistry*
  • Polylactic Acid-Polyglycolic Acid Copolymer
  • Solubility
  • Survivin
  • T-Lymphocytes, Cytotoxic / drug effects*
  • T-Lymphocytes, Cytotoxic / immunology
  • T-Lymphocytes, Cytotoxic / metabolism

Substances

  • BIRC5 protein, human
  • Cancer Vaccines
  • Delayed-Action Preparations
  • Inhibitor of Apoptosis Proteins
  • Lipopolysaccharides
  • MART-1 Antigen
  • MLANA protein, human
  • Peptide Fragments
  • Survivin
  • Polylactic Acid-Polyglycolic Acid Copolymer
  • Polyglycolic Acid
  • Lactic Acid