FAM13A, A Fatty Acid Oxidation Switch in Mitochondria. Friend or Foe in Chronic Obstructive Pulmonary Disease Pathogenesis?

Am J Respir Cell Mol Biol. 2017 Jun;56(6):689-691. doi: 10.1165/rcmb.2017-0080ED.

Authors

Gregory A Hawkins¹, Ana L Mora²

Affiliations

¹ 1 Center for Genomics and Personalized Medicine Research Wake Forest School of Medicine Winston-Salem, North Carolina.
² 2 Division of Pulmonary, Allergy, and Critical Care Medicine Vascular Medicine Institute University of Pittsburgh Pittsburgh, Pennsylvania.

No abstract available

Publication types

Editorial
Research Support, N.I.H., Extramural

MeSH terms

Alveolar Epithelial Cells / metabolism*
Alveolar Epithelial Cells / pathology
Animals
Fatty Acids / metabolism*
GTPase-Activating Proteins* / genetics
GTPase-Activating Proteins* / metabolism
Humans
Mitochondria* / genetics
Mitochondria* / metabolism
Mitochondria* / pathology
Oxidation-Reduction
Polymorphism, Genetic*
Pulmonary Disease, Chronic Obstructive* / genetics
Pulmonary Disease, Chronic Obstructive* / metabolism
Pulmonary Disease, Chronic Obstructive* / pathology

Substances

FAM13A protein, human
Fatty Acids
GTPase-Activating Proteins

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