Identification of monoclonal antibodies that inhibit the function of protein C inhibitor. Evidence for heparin-independent inhibition of activated protein C in plasma

Blood. 1988 Oct;72(4):1401-3.

Abstract

Monoclonal antibodies specific for protein C inhibitor (PCI) partially blocked the inactivation of activated protein C (APC) in plasma, whereas in a purified system, the PCI activity could be completely blocked. The inactivation of APC in normal and in PCI-depleted plasma was similar in the absence of heparin. The addition of heparin did not change the rate of inactivation of APC in PCI-depleted plasma, whereas in normal plasma a rapid phase of inhibition of APC was followed by a slower phase of inhibition. The slower phase was identical to the rate of inhibition of APC in the absence of heparin. After incubation of normal plasma with a monoclonal antibody specific for PCI that blocked its activity, there was no difference in heparin-dependent or heparin-independent inhibition of APC. These results indicate that in the absence of heparin PCI is unable to inactivate APC in a plasma environment.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibodies, Monoclonal / analysis*
  • Antibodies, Monoclonal / pharmacology
  • Blood Coagulation Tests
  • Blood Proteins / antagonists & inhibitors
  • Blood Proteins / immunology*
  • Heparin* / blood*
  • Humans
  • Mice
  • Mice, Inbred BALB C
  • Protease Inhibitors / analysis*
  • Protease Inhibitors / pharmacology
  • Protein C / blood
  • Protein C / immunology*
  • Protein C Inhibitor

Substances

  • Antibodies, Monoclonal
  • Blood Proteins
  • Protease Inhibitors
  • Protein C
  • Protein C Inhibitor
  • Heparin