Disruption of blood-nerve barriers in hereditary transthyretin (ATTR) amyloidosis

Amyloid. 2017 Mar;24(sup1):89-90. doi: 10.1080/13506129.2017.1278691.

Authors

Affiliations

¹ a Department of Neurology , Nagoya University Graduate School of Medicine , Nagoya , Japan.
² b Department of Medicine (Neurology and Rheumatology) , Shinshu University School of Medicine , Matsumoto , Japan , and.
³ c Department of Neurology , Graduate School of Medical Sciences, Kumamoto University , Kumamoto , Japan.

PMID: 28434331
DOI: 10.1080/13506129.2017.1278691

No abstract available

MeSH terms

Adult
Aged
Amyloid Neuropathies, Familial / metabolism*
Amyloid Neuropathies, Familial / pathology*
Benzoxazoles / pharmacology
Cell Survival / drug effects
Cell Survival / genetics
Female
Humans
Male
Middle Aged
Mutation / genetics
Plasma / cytology

Substances

Benzoxazoles
tafamidis

Supplementary concepts

Amyloidosis, Hereditary, Transthyretin-Related