Downregulation of miR-1 enhances tumorigenicity and invasiveness in oral squamous cell carcinomas

J Formos Med Assoc. 2017 Oct;116(10):782-789. doi: 10.1016/j.jfma.2016.12.003. Epub 2017 Jan 12.

Abstract

Background/purpose: Cumulative evidence suggest that microRNAs (miRNAs) function as biosignatures of oral squamous cell carcinomas (OSCC). However, the functional roles of miR-1 as well as its downstream targets in the regulation of tumorigenicity in OSCC remain unclear.

Methods: miRNAs RT-PCR analysis was performed to identify miR-1 as a putative candidate on mediating invasiveness of OSCC cells. Consequently, we elucidated the tumorigenicity of OSCC cells with miR-1 downregulation or overexpression, respectively. Finally, miR-1 on OSCC tumor tissues was examined.

Results: miR-1 levels were significantly downregulated in the malignant OSCC cells. Overexpression of miR-1 significantly reduced migration/invasiveness of OSCC cells. In addition, overexpression of miR-1 decreased cancer stem cells properties. Conversely, downregulation of miR-1 promotes migration and invasiveness in OSCC cells. We have shown that miR-1 is able to target Slug, suppressing their expression. Clinically, lower miR-1 expression was found in patients with advanced nodal metastasis OSCC.

Conclusion: miR-1 as novel biosignatures in OSCC lymph node metastatic patients, supporting the development of novel strategies for OSCC treatment.

Keywords: invasiveness; miR-1; oral squamous cell carcinomas.

MeSH terms

  • Carcinoma, Squamous Cell / genetics*
  • Carcinoma, Squamous Cell / pathology
  • Cell Line, Tumor
  • Down-Regulation
  • Female
  • Gene Expression Regulation, Neoplastic
  • Humans
  • Linear Models
  • Male
  • MicroRNAs / genetics*
  • Middle Aged
  • Mouth Neoplasms / genetics*
  • Mouth Neoplasms / pathology

Substances

  • MIRN1 microRNA, human
  • MicroRNAs