Abstract
A collaborative high throughput screen of 1.35 million compounds against mutant (R132H) isocitrate dehydrogenase IDH1 led to the identification of a novel series of inhibitors. Elucidation of the bound ligand crystal structure showed that the inhibitors exhibited a novel binding mode in a previously identified allosteric site of IDH1 (R132H). This information guided the optimization of the series yielding submicromolar enzyme inhibitors with promising cellular activity. Encouragingly, one compound from this series was found to induce myeloid differentiation in primary human IDH1 R132H AML cells in vitro.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Allosteric Regulation / drug effects
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Cell Differentiation / drug effects
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Dose-Response Relationship, Drug
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Drug Discovery*
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Enzyme Inhibitors / chemistry
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Enzyme Inhibitors / pharmacology*
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Humans
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Isocitrate Dehydrogenase / antagonists & inhibitors*
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Isocitrate Dehydrogenase / isolation & purification
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Isocitrate Dehydrogenase / metabolism
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Leukemia, Myeloid, Acute / enzymology*
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Leukemia, Myeloid, Acute / pathology
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Models, Molecular
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Molecular Structure
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Structure-Activity Relationship
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Tumor Cells, Cultured
Substances
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Enzyme Inhibitors
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Isocitrate Dehydrogenase
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IDH1 protein, human