Regulation of B-cell development and tolerance by different members of the miR-17∼92 family microRNAs

Nat Commun. 2016 Aug 2:7:12207. doi: 10.1038/ncomms12207.

Abstract

The molecular mechanisms that regulate B-cell development and tolerance remain incompletely understood. In this study, we identify a critical role for the miR-17∼92 microRNA cluster in regulating B-cell central tolerance and demonstrate that these miRNAs control early B-cell development in a cell-intrinsic manner. While the cluster member miR-19 suppresses the expression of Pten and plays a key role in regulating B-cell tolerance, miR-17 controls early B-cell development through other molecular pathways. These findings demonstrate differential control of two closely linked B-cell developmental stages by different members of a single microRNA cluster through distinct molecular pathways.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • B-Lymphocytes / physiology*
  • Cell Differentiation / genetics
  • Cells, Cultured
  • Female
  • Hematopoietic Stem Cell Transplantation
  • Hematopoietic Stem Cells / metabolism
  • Immune Tolerance / genetics*
  • Lymphocyte Activation / genetics*
  • Male
  • Mice, Inbred C57BL
  • Mice, Transgenic
  • MicroRNAs / physiology*
  • Models, Animal
  • PTEN Phosphohydrolase / genetics*

Substances

  • MIRN17-92 microRNA, mouse
  • MIRN19 microRNA, mouse
  • MicroRNAs
  • Mirn17 microRNA, mouse
  • PTEN Phosphohydrolase
  • Pten protein, mouse