Abstract
Two recent studies validate the LMNA-NTRK1 fusion as an oncogenic driver and therapeutic target of TRK inhibitors. The LMNA-NTRK1 fusion occurs at low frequency across multiple tumor types. The studies highlight the increasing need to develop molecular biomarker-based clinical trials across cancer subtypes.
©2016 American Association for Cancer Research.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Brain Neoplasms / metabolism*
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Brain Neoplasms / secondary
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Chemokine CCL2 / antagonists & inhibitors
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Chemokine CCL2 / metabolism
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Drug Resistance, Neoplasm* / drug effects
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Humans
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Organ Specificity
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PTEN Phosphohydrolase / deficiency*
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Protein Kinase Inhibitors / pharmacology*
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Protein Kinase Inhibitors / therapeutic use
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Small Molecule Libraries / pharmacology
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Small Molecule Libraries / therapeutic use
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Tumor Microenvironment
Substances
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CCL2 protein, human
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Chemokine CCL2
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Protein Kinase Inhibitors
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Small Molecule Libraries
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PTEN Phosphohydrolase
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PTEN protein, human