Microbiome as mediator: Do systemic infections start in the gut?

World J Gastroenterol. 2015 Oct 7;21(37):10487-92. doi: 10.3748/wjg.v21.i37.10487.

Abstract

The intestinal microbiome is emerging as a crucial mediator between external insults and systemic infections. New research suggests that our intestinal microorganisms contribute to critical illness and the development of non-gastrointestinal infectious diseases. Common pathways include a loss of fecal intestinal bacterial diversity and a disproportionate increase in toxogenic bacterial species. Therapeutic interventions targeting the microbiome - primarily probiotics - have yielded limited results to date. However, knowledge in this area is rapidly expanding and microbiome-based therapy such as short-chain fatty acids may eventually become a standard strategy for preventing systemic infections in the context of critical illness.

Keywords: Clostridium difficile; Critical illness; Gut; Microbiome; Probiotics; Short-chain fatty acids.

Publication types

  • Editorial
  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Clostridioides difficile
  • Clostridium Infections / microbiology
  • Clostridium Infections / therapy*
  • Critical Care
  • Critical Illness
  • Fatty Acids, Volatile / metabolism
  • Feces
  • Gastrointestinal Microbiome*
  • Humans
  • Intestines / microbiology*
  • Microbiota
  • Probiotics
  • Sepsis / microbiology

Substances

  • Fatty Acids, Volatile