Abstract
Directly modulating the activity of genome-editing proteins has the potential to increase their specificity by reducing activity following target locus modification. We developed Cas9 nucleases that are activated by the presence of a cell-permeable small molecule by inserting an evolved 4-hydroxytamoxifen-responsive intein at specific positions in Cas9. In human cells, conditionally active Cas9s modify target genomic sites with up to 25-fold higher specificity than wild-type Cas9.
Publication types
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Research Support, N.I.H., Extramural
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, Non-P.H.S.
MeSH terms
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Bacterial Proteins / drug effects
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Bacterial Proteins / genetics
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Cells, Cultured
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Endonucleases / drug effects
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Endonucleases / genetics*
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Genome / drug effects*
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HEK293 Cells
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High-Throughput Nucleotide Sequencing
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Humans
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Ligands
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Models, Molecular
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Protein Conformation
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Small Molecule Libraries
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Tamoxifen / analogs & derivatives
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Tamoxifen / pharmacology
Substances
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Bacterial Proteins
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Ligands
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Small Molecule Libraries
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Tamoxifen
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afimoxifene
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Endonucleases