A family of distal arthrogryposis type 5 due to a novel PIEZO2 mutation

Am J Med Genet A. 2015 May;167A(5):1100-6. doi: 10.1002/ajmg.a.36881. Epub 2015 Feb 25.

Abstract

Distal arthrogryposis (DA) encompasses a heterogeneous group of hereditary disorders with multiple congenital contractures predominant in the distal extremities. A total of 10 subtypes are proposed based on the pattern of contractures and association with extraarticular symptoms. DA5 is defined as a subtype with ptosis/oculomotor limitation. However, affected individuals have a variety of non-ocular features as well. We report on a two-generation family, including four affected individuals who all had congenital contractures of the distal joints, ptosis, restricted ocular movements, distinct facial appearance with deep-set eyes, and shortening of the 1st and 5th toes. The proband and her affected mother had restrictive lung disease, a recently recognized syndromic component of DA5, while younger patients did not. The proband had metacarpal and metatarsal synostosis, and the mother showed excavation of the optic disk. Whole-exome sequencing revealed a novel heterozygous mutation c.4456G>C (p.A1486P) of PIEZO2. PIEZO2 encodes a mechanosensitive ion channel, malfunction of which provides pleiotropic effects on joints, ocular muscles, lung function, and bone development.

Keywords: PIEZO2; distal arthrogryposis type 5; mechanosensitive ion channel; myopathy; optic nerve; restrictive lung disease; synostosis.

MeSH terms

  • Adolescent
  • Adult
  • Arthrogryposis / etiology
  • Arthrogryposis / genetics*
  • Arthrogryposis / physiopathology
  • Child
  • Exome / genetics
  • Female
  • Humans
  • Ion Channels / genetics*
  • Male
  • Muscular Diseases / genetics
  • Muscular Diseases / physiopathology
  • Mutation / genetics*
  • Optic Nerve / physiopathology*
  • Pedigree
  • Sequence Analysis, DNA
  • Synostosis / genetics
  • Synostosis / physiopathology

Substances

  • Ion Channels
  • PIEZO2 protein, human